Thomas A. Cooper

Affiliations: 
Department of Pathology and Immunology Baylor College of Medicine, Houston, TX 
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Publications

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Penna MS, Hu RC, Rodney GG, et al. (2023) The role of alternative splicing in skeletal muscle function. Life Science Alliance. 6
Rao AN, Campbell HM, Guan X, et al. (2021) Reversible cardiac disease features in an inducible CUG-repeat RNA expressing mouse model of myotonic dystrophy. Jci Insight
Johnson SJ, Cooper TA. (2020) Overlapping mechanisms of lncRNA and expanded microsatellite RNA. Wiley Interdisciplinary Reviews. Rna. e1634
Cox DC, Guan X, Xia Z, et al. (2020) Increased nuclear but not cytoplasmic activities of CELF1 protein leads to muscle wasting. Human Molecular Genetics
Misra C, Bangru S, Lin F, et al. (2020) Aberrant Expression of a Non-muscle RBFOX2 Isoform Triggers Cardiac Conduction Defects in Myotonic Dystrophy. Developmental Cell
Sharp L, Cox DC, Cooper TA. (2019) Endurance exercise leads to beneficial molecular and physiological effects in a mouse model of myotonic dystrophy type 1. Muscle & Nerve
Wang ET, Treacy D, Eichinger K, et al. (2018) Transcriptome alterations in myotonic dystrophy skeletal muscle and heart. Human Molecular Genetics
Payer LM, Steranka JP, Ardeljan D, et al. (2018) Alu insertion variants alter mRNA splicing. Nucleic Acids Research
Pang PD, Alsina KM, Cao S, et al. (2018) CRISPR -Mediated Expression of the Fetal Scn5a Isoform in Adult Mice Causes Conduction Defects and Arrhythmias. Journal of the American Heart Association. 7: e010393
Eric Blue R, Koushik A, Engels NM, et al. (2018) Modulation of alternative splicing of trafficking genes by genome editing reveals functional consequences in muscle biology. The International Journal of Biochemistry & Cell Biology
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