Thomas A. Cooper
Affiliations: | Department of Pathology and Immunology | Baylor College of Medicine, Houston, TX |
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Publications
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| Nitschke L, Hu RC, Miller AN, et al. (2024) Rescue of Scn5a mis-splicing does not improve the structural and functional heart defects of a DM1 heart mouse model. Human Molecular Genetics |
| Penna MS, Hu RC, Rodney GG, et al. (2023) The role of alternative splicing in skeletal muscle function. Life Science Alliance. 6 |
| Rao AN, Campbell HM, Guan X, et al. (2021) Reversible cardiac disease features in an inducible CUG-repeat RNA expressing mouse model of myotonic dystrophy. Jci Insight |
| Johnson SJ, Cooper TA. (2020) Overlapping mechanisms of lncRNA and expanded microsatellite RNA. Wiley Interdisciplinary Reviews. Rna. e1634 |
| Cox DC, Guan X, Xia Z, et al. (2020) Increased nuclear but not cytoplasmic activities of CELF1 protein leads to muscle wasting. Human Molecular Genetics |
| Misra C, Bangru S, Lin F, et al. (2020) Aberrant Expression of a Non-muscle RBFOX2 Isoform Triggers Cardiac Conduction Defects in Myotonic Dystrophy. Developmental Cell |
| Sharp L, Cox DC, Cooper TA. (2019) Endurance exercise leads to beneficial molecular and physiological effects in a mouse model of myotonic dystrophy type 1. Muscle & Nerve |
| Wang ET, Treacy D, Eichinger K, et al. (2018) Transcriptome alterations in myotonic dystrophy skeletal muscle and heart. Human Molecular Genetics |
| Payer LM, Steranka JP, Ardeljan D, et al. (2018) Alu insertion variants alter mRNA splicing. Nucleic Acids Research |
| Pang PD, Alsina KM, Cao S, et al. (2018) CRISPR -Mediated Expression of the Fetal Scn5a Isoform in Adult Mice Causes Conduction Defects and Arrhythmias. Journal of the American Heart Association. 7: e010393 |