Chunyan Ren
Affiliations: | Mount Sinai School of Medicine, New York, NY |
Area:
Biophysics and Systems PharmacologyGoogle:
"Chunyan Ren"Mean distance: (not calculated yet)
Parents
Sign in to add mentor| Ming-Ming Zhou | grad student | 2009-2015 | Mount Sinai | |
| (Structure-based design of epigenetic chemical modulators) | ||||
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Publications
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| Liu N, Konuma T, Sharma R, et al. (2023) Histone H3 lysine 27 crotonylation mediates gene transcriptional repression in chromatin. Molecular Cell |
| Cheung KL, Jaganathan A, Hu Y, et al. (2022) HIPK2 directs cell type-specific regulation of STAT3 transcriptional activity in Th17 cell differentiation. Proceedings of the National Academy of Sciences of the United States of America. 119: e2117112119 |
| Vinjamur DS, Yao Q, Cole MA, et al. (2021) ZNF410 represses fetal globin by singular control of CHD4. Nature Genetics |
| Zeng J, Wu Y, Ren C, et al. (2020) Therapeutic base editing of human hematopoietic stem cells. Nature Medicine. 26: 535-541 |
| Ma Y, Xu B, Yu J, et al. (2020) Fli-1 Activation through Targeted Promoter Activity Regulation Using a Novel 3', 5'-diprenylated Chalcone Inhibits Growth and Metastasis of Prostate Cancer Cells. International Journal of Molecular Sciences. 21 |
| Kim DH, Ren C, Ryou C, et al. (2019) Direct interaction of DNMT inhibitors to PrP suppresses pathogenic process of prion. Acta Pharmaceutica Sinica. B. 9: 952-959 |
| Sher F, Hossain M, Seruggia D, et al. (2019) Rational targeting of a NuRD subcomplex guided by comprehensive in situ mutagenesis. Nature Genetics |
| Wu Y, Zeng J, Roscoe BP, et al. (2019) Highly efficient therapeutic gene editing of human hematopoietic stem cells. Nature Medicine |
| Ren C, Zhang G, Han F, et al. (2018) Spatially constrained tandem bromodomain inhibition bolsters sustained repression of BRD4 transcriptional activity for TNBC cell growth. Proceedings of the National Academy of Sciences of the United States of America |
| Konuma T, Yu D, Zhao C, et al. (2017) Structural Mechanism of the Oxygenase JMJD6 Recognition by the Extraterminal (ET) Domain of BRD4. Scientific Reports. 7: 16272 |