Sai Prasad Pydi

Affiliations: 
National Institutes of Health, Bethesda, MD 
Google:
"Sai Pydi"
Mean distance: (not calculated yet)
 
BETA: Related publications

Publications

You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect.

Barella LF, Jain S, Pydi SP. (2022) G protein-coupled receptors: Role in metabolic disorders. Frontiers in Endocrinology. 13: 984253
Meister J, Wang L, Pydi SP, et al. (2021) Chemogenetic approaches to identify metabolically important GPCR signaling pathways: therapeutic implications. Journal of Neurochemistry
Kimura T, Pydi SP, Pham J, et al. (2020) Metabolic Functions of G Protein-Coupled Receptors in Hepatocytes-Potential Applications for Diabetes and NAFLD. Biomolecules. 10
Wang L, Zhu L, Meister J, et al. (2020) Use of DREADD Technology to Identify Novel Targets for Antidiabetic Drugs. Annual Review of Pharmacology and Toxicology
Pydi SP, Jain S, Barella LF, et al. (2020) β-arrestin-1 suppresses myogenic reprogramming of brown fat to maintain euglycemia. Science Advances. 6: eaba1733
Wang L, Pydi SP, Zhu L, et al. (2020) Adipocyte G signaling is essential for maintaining whole-body glucose homeostasis and insulin sensitivity. Nature Communications. 11: 2995
Dattaroy D, Barella LF, Pydi SP, et al. (2020) 2129-P: Glucagon Secretion from Pancreatic Islets Is Regulated by Beta-Arrestin-1 Diabetes. 69
Pydi SP, Jain S, Barella LF, et al. (2020) 1694-P: Beta-Arrestin 1 Suppresses Myogenic Reprogramming of Brown Fat to Maintain Euglycemia Diabetes. 69
Wang L, Pydi SP, Cui Y, et al. (2019) Selective activation of G signaling in adipocytes causes striking metabolic improvements in mice. Molecular Metabolism
Pydi SP, Jain S, Tung W, et al. (2019) Adipocyte β-arrestin-2 is essential for maintaining whole body glucose and energy homeostasis. Nature Communications. 10: 2936
See more...