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Gary Yellen

Affiliations: 
Neurobiology Harvard Medical School, Boston, MA, United States 
Area:
Ion Channel Biophysics, Metabolism and Excitability
Website:
http://www.fas.harvard.edu/~biophys/Gary_Yellen.htm
Google:
"Gary Yellen"
Bio:

http://yellen.hms.harvard.edu/
http://dms.hms.harvard.edu/neuroscience/fac/yellen.php

Mean distance: 13.02 (cluster 46)
 
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Cross-listing: Neurotree

Parents

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Charles F. Stevens grad student 1979-1984 Yale School of Medicine (Neurotree)
Christopher Miller post-doc 1984-1986 Brandeis

Children

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Ki Soon Shin grad student Harvard (Neurotree)
Andrew Lutas grad student 2010- Harvard Medical School (Neurotree)
Sarah Webster grad student 1997-2004 Harvard (Neurotree)
John Paul Dekker grad student 2006 Harvard (Neurotree)
James Berg grad student 2008 Harvard
Geoffrey Richard Tanner grad student 2009 Harvard (Neurotree)
Yin Pun Hung grad student 2012 Harvard (Neurotree)
Yin P. (Rex) Hung grad student 2008-2012 Harvard Medical School (Neurotree)
Donato del Camino post-doc Harvard Medical School (Neurotree)
Miguel Holmgren post-doc Harvard Medical School (Neurotree)
Sujung Ryu post-doc Harvard Medical School (Neurotree)
Juan-Ramón Martínez-François post-doc 2010- Harvard Medical School (Neurotree)
Rebecca A. Mongeon post-doc 2011- Harvard Medical School (Neurotree)
Carlos Manlio Díaz García post-doc 2014- Harvard Medical School (Neurotree)
Dorothy M. Koveal post-doc 2016- Harvard Medical School (Neurotree)
Brad S. Rothberg post-doc 1999-2002 Harvard Medical School
Catherine Proenza post-doc 2002-2005 Harvard Medical School
Mathew C. Tantama post-doc 2008-2014 Harvard Medical School
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Publications

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Díaz-García CM, Yellen G. (2018) Neurons rely on glucose rather than astrocytic lactate during stimulation. Journal of Neuroscience Research
Martínez-François JR, Fernández-Agüera MC, Nathwani N, et al. (2018) BAD and KATP channels regulate neuron excitability and epileptiform activity. Elife. 7
Hung YP, Teragawa C, Kosaisawe N, et al. (2017) Akt regulation of glycolysis mediates bioenergetic stability in epithelial cells. Elife. 6
Foley J, Burnham V, Tedoldi M, et al. (2017) BAD knockout provides metabolic seizure resistance in a genetic model of epilepsy with sudden unexplained death in epilepsy. Epilepsia
Masia R, McCarty WJ, Lahmann C, et al. (2017) Live cell imaging of cytosolic NADH/NAD(+) ratio in hepatocytes and liver slices. American Journal of Physiology. Gastrointestinal and Liver Physiology. ajpgi.00093.2017
Díaz-García CM, Mongeon R, Lahmann C, et al. (2017) Neuronal Stimulation Triggers Neuronal Glycolysis and Not Lactate Uptake. Cell Metabolism. 26: 361-374.e4
Lutas A, Lahmann C, Soumillon M, et al. (2016) The leak channel NALCN controls tonic firing and glycolytic sensitivity of substantia nigra pars reticulata neurons. Elife. 5
Chen Y, Saulnier JL, Yellen G, et al. (2016) Corrigendum: A PKA activity sensor for quantitative analysis of endogenous GPCR signaling via 2-photon FRET-FLIM imaging. Frontiers in Pharmacology. 7: 46
Mongeon R, Venkatachalam V, Yellen G. (2016) Cytosolic NADH-NAD+ redox visualized in brain slices by two-photon fluorescence lifetime biosensor imaging. Antioxidants & Redox Signaling
Schoeler NE, Leu C, White J, et al. (2015) Variants in KCNJ11 and BAD do not predict response to ketogenic dietary therapies for epilepsy. Epilepsy Research. 118: 22-28
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