Alan M. Weiner, Ph.D.
Affiliations: | Biochemistry | University of Washington, Seattle, Seattle, WA |
Area:
the origin of lifeWebsite:
https://depts.washington.edu/biowww/pages/faculty-Weiner.shtmlGoogle:
"Alan M. Weiner"Mean distance: 9.51 | S | N | B | C | P |
Parents
Sign in to add mentor| Klaus K. Weber | grad student | 1973 | Harvard | |
| (Natural read-through in coliphage Qβ) | ||||
| Arthur Kornberg | post-doc | 1973-1974 | Stanford Medical School | |
| Harvey F. Lodish | post-doc | 1974-1976 | MIT | |
Children
Sign in to add trainee| Jo Ann Wise | grad student | 1981 | Yale (PombeTree) |
| Hua-Ying Fan | grad student | 2000 | University of Washington |
| John C. Newman | grad student | 2002-2006 | University of Washington (Neurotree) |
| Lucas Gray | grad student | 2012 | University of Washington |
| Kozo Tomita | post-doc | 1999-2002 | University of Washington |
BETA: Related publications
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Publications
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| Pavelitz T, Gray LT, Padilla SL, et al. (2013) PGBD5: a neural-specific intron-containing piggyBac transposase domesticated over 500 million years ago and conserved from cephalochordates to humans. Mobile Dna. 4: 23 |
| Weiner AM, Gray LT. (2013) What role (if any) does the highly conserved CSB-PGBD3 fusion protein play in Cockayne syndrome? Mechanisms of Ageing and Development. 134: 225-33 |
| Gray LT, Fong KK, Pavelitz T, et al. (2012) Tethering of the conserved piggyBac transposase fusion protein CSB-PGBD3 to chromosomal AP-1 proteins regulates expression of nearby genes in humans. Plos Genetics. 8: e1002972 |
| Bailey AD, Gray LT, Pavelitz T, et al. (2012) The conserved Cockayne syndrome B-piggyBac fusion protein (CSB-PGBD3) affects DNA repair and induces both interferon-like and innate antiviral responses in CSB-null cells. Dna Repair. 11: 488-501 |
| Gray LT, Weiner AM. (2010) Ubiquitin recognition by the Cockayne syndrome group B protein: binding will set you free. Molecular Cell. 38: 621-2 |
| Cho HD, Sood VD, Baker D, et al. (2008) On the role of a conserved, potentially helix-breaking residue in the tRNA-binding alpha-helix of archaeal CCA-adding enzymes. Rna (New York, N.Y.). 14: 1284-9 |
| Pavelitz T, Bailey AD, Elco CP, et al. (2008) Human U2 snRNA genes exhibit a persistently open transcriptional state and promoter disassembly at metaphase. Molecular and Cellular Biology. 28: 3573-88 |
| Newman JC, Bailey AD, Fan HY, et al. (2008) An abundant evolutionarily conserved CSB-PiggyBac fusion protein expressed in Cockayne syndrome. Plos Genetics. 4: e1000031 |
| Cho HD, Verlinde CL, Weiner AM. (2007) Reengineering CCA-adding enzymes to function as (U,G)- or dCdCdA-adding enzymes or poly(C,A) and poly(U,G) polymerases. Proceedings of the National Academy of Sciences of the United States of America. 104: 54-9 |
| Newman JC, Bailey AD, Weiner AM. (2006) Cockayne syndrome group B protein (CSB) plays a general role in chromatin maintenance and remodeling. Proceedings of the National Academy of Sciences of the United States of America. 103: 9613-8 |