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David Szuts

Affiliations: 
Institute of Enzymology Research Centre for Natural Sciences, Budapest 
Area:
DNA repair
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Parents

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Mariann Bienz grad student 1995-1999 (FlyTree)
Torsten Krude post-doc 1999-2004 Cambridge
Julian E. Sale post-doc 2004-2008 MRC LMB

Children

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Publications

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Lózsa R, Németh E, Gervai JZ, et al. (2023) DNA mismatch repair protects the genome from oxygen-induced replicative mutagenesis. Nucleic Acids Research
Gyüre Z, Póti Á, Németh E, et al. (2023) Spontaneous mutagenesis in human cells is controlled by REV1-Polymerase ζ and PRIMPOL. Cell Reports. 42: 112887
Póti Á, Szikriszt B, Gervai JZ, et al. (2022) Characterisation of the spectrum and genetic dependence of collateral mutations induced by translesion DNA synthesis. Plos Genetics. 18: e1010051
Chen D, Gervai JZ, Póti Á, et al. (2022) BRCA1 deficiency specific base substitution mutagenesis is dependent on translesion synthesis and regulated by 53BP1. Nature Communications. 13: 226
Szeltner Z, Póti Á, Harami GM, et al. (2021) Evaluation and modulation of DNA lesion bypass in an SV40 large T antigen-based in vitro replication system. Febs Open Bio
Szikriszt B, Póti Á, Németh E, et al. (2021) A comparative analysis of the mutagenicity of platinum-containing chemotherapeutic agents reveals direct and indirect mutagenic mechanisms. Mutagenesis. 36: 75-86
Pálinkás HL, Békési A, Róna G, et al. (2020) Genome-wide alterations of uracil distribution patterns in human DNA upon chemotherapeutic treatments. Elife. 9
Póti Á, Gyergyák H, Németh E, et al. (2019) Correlation of homologous recombination deficiency induced mutational signatures with sensitivity to PARP inhibitors and cytotoxic agents. Genome Biology. 20: 240
Vető B, Szabó P, Bacquet C, et al. (2018) Inhibition of DNA methyltransferase leads to increased genomic 5-hydroxymethylcytosine levels in hematopoietic cells. Febs Open Bio. 8: 584-592
Gervai JZ, Gálicza J, Szeltner Z, et al. (2017) A genetic study based on PCNA-ubiquitin fusions reveals no requirement for PCNA polyubiquitylation in DNA damage tolerance. Dna Repair. 54: 46-54
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