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| Al-Zaid B, Chacko S, Ezeamuzie CI, et al. (2022) Differential effects of glucose-dependent insulinotropic polypeptide receptor/glucagon-like peptide-1 receptor heteromerization on cell signaling when expressed in HEK-293 cells. Pharmacology Research & Perspectives. 10: e01013 |
| Ismaeil A, Babiker F, Al-Sabah S. (2022) Discrepancy between the Actions of Glucagon-like Peptide-1 Receptor Ligands in the Protection of the Heart against Ischemia Reperfusion Injury. Pharmaceuticals (Basel, Switzerland). 15 |
| Al-Sabah S, Adi L, Bünemann M, et al. (2020) The Effect of Cell Surface Expression and Linker Sequence on the Recruitment of Arrestin to the GIP Receptor. Frontiers in Pharmacology. 11: 1271 |
| Al-Zamel N, Al-Sabah S, Luqmani Y, et al. (2020) Correction: Al-Zamel, N., et al. A Dual GLP-1/GIP Receptor Agonist Does Not Antagonize Glucagon at Its Receptor but May Act as a Biased Agonist at the GLP-1 Receptor. 2019, , 3532. International Journal of Molecular Sciences. 21 |
| Al-Zamel N, Al-Sabah S, Luqmani Y, et al. (2019) A Dual GLP-1/GIP Receptor Agonist Does Not Antagonize Glucagon at Its Receptor but May Act as a Biased Agonist at the GLP-1 Receptor. International Journal of Molecular Sciences. 20 |
| Shaaban G, Oriowo M, Al-Sabah S. (2016) Rate of Homologous Desensitization and Internalization of the GLP-1 Receptor. Molecules (Basel, Switzerland). 22 |
| Al-Sabah S. (2015) Molecular Pharmacology of the Incretin Receptors. Medical Principles and Practice : International Journal of the Kuwait University, Health Science Centre. 15-21 |
| Zindel D, Butcher AJ, Al-Sabah S, et al. (2015) Engineered hyperphosphorylation of the β2-adrenoceptor prolongs arrestin-3 binding and induces arrestin internalization. Molecular Pharmacology. 87: 349-62 |
| Al-Sabah S, Al-Fulaij M, Shaaban G, et al. (2014) The GIP receptor displays higher basal activity than the GLP-1 receptor but does not recruit GRK2 or arrestin3 effectively. Plos One. 9: e106890 |
| Lampe M, Pierre F, Al-Sabah S, et al. (2014) Dual single-scission event analysis of constitutive transferrin receptor (TfR) endocytosis and ligand-triggered β2-adrenergic receptor (β2AR) or Mu-opioid receptor (MOR) endocytosis. Molecular Biology of the Cell. 25: 3070-80 |