Year |
Citation |
Score |
2015 |
Acosta-Alvear D, Cho MY, Wild T, Buchholz TJ, Lerner AG, Simakova O, Hahn J, Korde N, Landgren O, Maric I, Choudhary C, Walter P, Weissman JS, Kampmann M. Paradoxical resistance of multiple myeloma to proteasome inhibitors by decreased levels of 19S proteasomal subunits. Elife. 4. PMID 26327694 DOI: 10.7554/Elife.08153 |
0.464 |
|
2015 |
Acosta-Alvear D, Cho MY, Wild T, Buchholz TJ, Lerner AG, Simakova O, Hahn J, Korde N, Landgren O, Maric I, Choudhary C, Walter P, Weissman JS, Kampmann M. Author response: Paradoxical resistance of multiple myeloma to proteasome inhibitors by decreased levels of 19S proteasomal subunits Elife. DOI: 10.7554/Elife.08153.019 |
0.463 |
|
2015 |
Masto VB, Lerner AG, Arastu-Kapur S. Abstract 1735: Identification of novel synthetic lethal interactions in small cell lung cancer with the proteasome inhibitor carfilzomib Cancer Research. 75: 1735-1735. DOI: 10.1158/1538-7445.Am2015-1735 |
0.365 |
|
2013 |
Lin T, Lerner A, Kirk CJ, Arastu-Kapur S. Survival Pathways Stabilized By Proteasome Inhibition Reveal PIM2 As a Novel Target For Potentiating The Anti-Myeloma Activity Of Carfilzomib Blood. 122: 4440-4440. DOI: 10.1182/Blood.V122.21.4440.4440 |
0.463 |
|
2013 |
Lowe E, Maglathlin R, Lerner A, Taunton J, Kirk CJ, Arastu-Kapur S. Blocking Protein Secretion and Degradation Is a Novel Treatment Strategy For Malignant Cells With High Protein Load Blood. 122: 4439-4439. DOI: 10.1182/Blood.V122.21.4439.4439 |
0.4 |
|
2012 |
Lerner AG, Upton JP, Praveen PV, Ghosh R, Nakagawa Y, Igbaria A, Shen S, Nguyen V, Backes BJ, Heiman M, Heintz N, Greengard P, Hui S, Tang Q, Trusina A, et al. IRE1α induces thioredoxin-interacting protein to activate the NLRP3 inflammasome and promote programmed cell death under irremediable ER stress. Cell Metabolism. 16: 250-64. PMID 22883233 DOI: 10.1016/J.Cmet.2012.07.007 |
0.478 |
|
2009 |
Han D, Lerner AG, Vande Walle L, Upton JP, Xu W, Hagen A, Backes BJ, Oakes SA, Papa FR. IRE1alpha kinase activation modes control alternate endoribonuclease outputs to determine divergent cell fates. Cell. 138: 562-75. PMID 19665977 DOI: 10.1016/J.Cell.2009.07.017 |
0.473 |
|
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