| Year |
Citation |
Score |
| 2023 |
Li AJ, Wang Q, Rogers RC, Hermann G, Ritter RC, Ritter S. Chemogenetic activation of ventral medullary astrocytes enhances feeding and corticosterone release in response to mild glucoprivation. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. PMID 37424401 DOI: 10.1152/ajpregu.00079.2023 |
0.316 |
|
| 2019 |
Rogers RC, Burke SJ, Collier JJ, Ritter S, Hermann GE. Evidence that hindbrain astrocytes in the rat detect low glucose with a glucose transporter 2-phospholipase C-calcium release mechanism. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. PMID 31596114 DOI: 10.1152/Ajpregu.00133.2019 |
0.329 |
|
| 2019 |
Seamon M, Ahn W, Li AJ, Ritter S, Harris RBS. Leptin receptor-expressing neurons in the ventromedial nucleus of the hypothalamus contribute to weight loss caused by fourth ventricle leptin infusions. American Journal of Physiology. Endocrinology and Metabolism. PMID 31361549 DOI: 10.1152/Ajpendo.00205.2019 |
0.477 |
|
| 2019 |
Ritter S, Li AJ, Wang Q. Hindbrain glucoregulatory mechanisms: Critical role of catecholamine neurons in the ventrolateral medulla. Physiology & Behavior. 112568. PMID 31173784 DOI: 10.1016/J.Physbeh.2019.112568 |
0.369 |
|
| 2018 |
Li AJ, Wang Q, Ritter S. Activation of catecholamine neurons in the ventral medulla reduces CCK-induced hypophagia and c-Fos activation in dorsal medullary catecholamine neurons. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. PMID 29874094 DOI: 10.1152/Ajpregu.00107.2018 |
0.432 |
|
| 2018 |
Rogers RC, McDougal DH, Ritter S, Qualls-Creekmore E, Hermann GE. Response of catecholaminergic neurons in the mouse hindbrain to glucoprivic stimuli is astrocyte dependent. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. PMID 29590557 DOI: 10.1152/Ajpregu.00368.2017 |
0.369 |
|
| 2017 |
Li AJ, Wang Q, Ritter S. Selective pharmacogenetic activation of catecholamine subgroups in the ventrolateral medulla elicits key glucoregulatory responses. Endocrinology. PMID 29077837 DOI: 10.1210/En.2017-00630 |
0.379 |
|
| 2016 |
Rogers RC, Ritter S, Hermann GE. Hindbrain cytoglucopenia-induced increases in systemic blood glucose levels by 2-deoxyglucose depend on intact astrocytes and adenosine release. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. ajpregu.00493.2015. PMID 27101298 DOI: 10.1152/Ajpregu.00493.2015 |
0.393 |
|
| 2016 |
Li AJ, Wiater MF, Wang Q, Wank SA, Ritter S. Deletion of GPR40 Fatty Acid Receptor Gene in Mice Blocks Mercaptoacetate-Induced Feeding. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. ajpregu.00548.2015. PMID 26984894 DOI: 10.1152/Ajpregu.00548.2015 |
0.79 |
|
| 2016 |
Li AJ, Wang Q, Dinh TT, Simasko SM, Ritter S. Mercaptoacetate blocks fatty acid-induced GLP-1 secretion in male rats by directly antagonizing GPR40 fatty acid receptors. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. ajpregu.00387.2015. PMID 26791830 DOI: 10.1152/Ajpregu.00387.2015 |
0.431 |
|
| 2015 |
Li AJ, Wang Q, Davis H, Wang R, Ritter S. Orexin-A Enhances Feeding in Male Rats by Activating Hindbrain Catecholamine Neurons. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. ajpregu.00065.2015. PMID 26062632 DOI: 10.1152/Ajpregu.00065.2015 |
0.487 |
|
| 2015 |
Li AJ, Wang Q, Elsarelli MM, Brown RL, Ritter S. Hindbrain Catecholamine Neurons Activate Orexin Neurons During Systemic Glucoprivation in Male Rats. Endocrinology. 156: 2807-20. PMID 25978516 DOI: 10.1210/En.2015-1138 |
0.454 |
|
| 2014 |
Darling RA, Zhao H, Kinch D, Li AJ, Simasko SM, Ritter S. Mercaptoacetate and fatty acids exert direct and antagonistic effects on nodose neurons via GPR40 fatty acid receptors. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 307: R35-43. PMID 24760994 DOI: 10.1152/Ajpregu.00536.2013 |
0.778 |
|
| 2014 |
Li AJ, Wang Q, Dinh TT, Powers BR, Ritter S. Stimulation of feeding by three different glucose-sensing mechanisms requires hindbrain catecholamine neurons. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 306: R257-64. PMID 24381177 DOI: 10.1152/Ajpregu.00451.2013 |
0.502 |
|
| 2013 |
Li AJ, Wang Q, Dinh TT, Wiater MF, Eskelsen AK, Ritter S. Hindbrain catecholamine neurons control rapid switching of metabolic substrate use during glucoprivation in male rats. Endocrinology. 154: 4570-9. PMID 24064356 DOI: 10.1210/En.2013-1589 |
0.806 |
|
| 2013 |
Wiater MF, Li AJ, Dinh TT, Jansen HT, Ritter S. Leptin-sensitive neurons in the arcuate nucleus integrate activity and temperature circadian rhythms and anticipatory responses to food restriction. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 305: R949-60. PMID 23986359 DOI: 10.1152/Ajpregu.00032.2013 |
0.787 |
|
| 2012 |
Li AJ, Wiater MF, Oostrom MT, Smith BR, Wang Q, Dinh TT, Roberts BL, Jansen HT, Ritter S. Leptin-sensitive neurons in the arcuate nuclei contribute to endogenous feeding rhythms. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 302: R1313-26. PMID 22492818 DOI: 10.1152/Ajpregu.00086.2012 |
0.793 |
|
| 2011 |
Wiater MF, Mukherjee S, Li AJ, Dinh TT, Rooney EM, Simasko SM, Ritter S. Circadian integration of sleep-wake and feeding requires NPY receptor-expressing neurons in the mediobasal hypothalamus. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 301: R1569-83. PMID 21880863 DOI: 10.1152/Ajpregu.00168.2011 |
0.77 |
|
| 2011 |
Ritter S, Li AJ, Wang Q, Dinh TT. Minireview: The value of looking backward: the essential role of the hindbrain in counterregulatory responses to glucose deficit. Endocrinology. 152: 4019-32. PMID 21878511 DOI: 10.1210/En.2010-1458 |
0.39 |
|
| 2011 |
Li AJ, Wang Q, Ritter S. Participation of hindbrain AMP-activated protein kinase in glucoprivic feeding. Diabetes. 60: 436-42. PMID 21270255 DOI: 10.2337/db10-0352 |
0.392 |
|
| 2010 |
Emanuel AJ, Ritter S. Hindbrain catecholamine neurons modulate the growth hormone but not the feeding response to ghrelin. Endocrinology. 151: 3237-46. PMID 20463049 DOI: 10.1210/En.2010-0219 |
0.797 |
|
| 2010 |
Hudson BD, Emanuel AJ, Wiater MF, Ritter S. The lipoprivic control of feeding is governed by fat metabolism, not by leptin or adipose depletion. Endocrinology. 151: 2087-96. PMID 20203155 DOI: 10.1210/En.2009-1043 |
0.774 |
|
| 2010 |
Wu X, Wiater MF, Ritter S. NPAS2 deletion impairs responses to restricted feeding but not to metabolic challenges. Physiology & Behavior. 99: 466-71. PMID 20026146 DOI: 10.1016/J.Physbeh.2009.12.010 |
0.804 |
|
| 2009 |
Darling RA, Ritter S. 2-Deoxy-D-glucose, but not mercaptoacetate, increases food intake in decerebrate rats. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 297: R382-6. PMID 19494173 DOI: 10.1152/Ajpregu.90827.2008 |
0.818 |
|
| 2009 |
Li AJ, Wang Q, Dinh TT, Ritter S. Simultaneous silencing of Npy and Dbh expression in hindbrain A1/C1 catecholamine cells suppresses glucoprivic feeding. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 29: 280-7. PMID 19129404 DOI: 10.1523/Jneurosci.4267-08.2009 |
0.42 |
|
| 2009 |
Wiater M, Ritter S. β-Mercaptoacetate-induced feeding is not altered by endogenous, central or peripheral leptin Appetite. 52: 865. DOI: 10.1016/J.Appet.2009.04.208 |
0.759 |
|
| 2009 |
Emanuel A, Dinh T, Ritter S. Hindbrain catecholamine neurons contribute to the growth hormone but not the feeding response to ghrelin Appetite. 52: 829. DOI: 10.1016/J.Appet.2009.04.060 |
0.368 |
|
| 2008 |
Li AJ, Dinh TT, Ritter S. Hyperphagia and obesity produced by arcuate injection of NPY-saporin do not require upregulation of lateral hypothalamic orexigenic peptide genes. Peptides. 29: 1732-9. PMID 18577407 DOI: 10.1016/J.Peptides.2008.05.026 |
0.442 |
|
| 2007 |
Wiater MF, Hudson BD, Virgin Y, Ritter S. Protein appetite is increased after central leptin-induced fat depletion. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 293: R1468-73. PMID 17596325 DOI: 10.1152/Ajpregu.00322.2007 |
0.803 |
|
| 2007 |
Taylor K, Lester E, Hudson B, Ritter S. Hypothalamic and hindbrain NPY, AGRP and NE increase consummatory feeding responses. Physiology & Behavior. 90: 744-50. PMID 17289093 DOI: 10.1016/J.Physbeh.2006.12.014 |
0.455 |
|
| 2007 |
Andrew SF, Dinh TT, Ritter S. Localized glucoprivation of hindbrain sites elicits corticosterone and glucagon secretion. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 292: R1792-8. PMID 17218439 DOI: 10.1152/Ajpregu.00777.2006 |
0.36 |
|
| 2007 |
Hudson B, Wiater M, Ritter S. Chronic central leptin treatment increases protein intake in rats. Appetite. 49: 299. DOI: 10.1016/J.Appet.2007.03.095 |
0.795 |
|
| 2007 |
Hudson B, Wiater M, Ritter S. Hindbrain catecholamine neurons contribute to control of daily food intake during chronic leptin treatment. Appetite. 49: 299. DOI: 10.1016/J.Appet.2007.03.093 |
0.818 |
|
| 2006 |
Ritter S, Dinh TT, Li AJ. Hindbrain catecholamine neurons control multiple glucoregulatory responses. Physiology & Behavior. 89: 490-500. PMID 16887153 DOI: 10.1016/J.Physbeh.2006.05.036 |
0.454 |
|
| 2006 |
Dinh TT, Flynn FW, Ritter S. Hypotensive hypovolemia and hypoglycemia activate different hindbrain catecholamine neurons with projections to the hypothalamus. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 291: R870-9. PMID 16675637 DOI: 10.1152/Ajpregu.00094.2006 |
0.486 |
|
| 2006 |
Li AJ, Wang Q, Ritter S. Differential responsiveness of dopamine-beta-hydroxylase gene expression to glucoprivation in different catecholamine cell groups. Endocrinology. 147: 3428-34. PMID 16614076 DOI: 10.1210/En.2006-0235 |
0.381 |
|
| 2005 |
Bugarith K, Dinh TT, Li AJ, Speth RC, Ritter S. Basomedial hypothalamic injections of neuropeptide Y conjugated to saporin selectively disrupt hypothalamic controls of food intake. Endocrinology. 146: 1179-91. PMID 15604214 DOI: 10.1210/En.2004-1166 |
0.812 |
|
| 2005 |
Ritter S, Dinh TT, Bugarith K, Salter DM. Chemical dissection of brain glucoregulatory circuitry Molecular Neurosurgery With Targeted Toxins. 181-218. DOI: 10.1007/978-1-59259-896-0_8 |
0.767 |
|
| 2004 |
Hudson B, Ritter S. Hindbrain catecholamine neurons mediate consummatory responses to glucoprivation. Physiology & Behavior. 82: 241-50. PMID 15276785 DOI: 10.1016/J.Physbeh.2004.03.032 |
0.529 |
|
| 2004 |
Li AJ, Ritter S. Glucoprivation increases expression of neuropeptide Y mRNA in hindbrain neurons that innervate the hypothalamus. The European Journal of Neuroscience. 19: 2147-54. PMID 15090041 DOI: 10.1111/J.1460-9568.2004.03287.X |
0.415 |
|
| 2003 |
I'Anson H, Sundling LA, Roland SM, Ritter S. Immunotoxic destruction of distinct catecholaminergic neuron populations disrupts the reproductive response to glucoprivation in female rats. Endocrinology. 144: 4325-31. PMID 12960045 DOI: 10.1210/En.2003-0258 |
0.462 |
|
| 2003 |
Ritter S, Watts AG, Dinh TT, Sanchez-Watts G, Pedrow C. Immunotoxin lesion of hypothalamically projecting norepinephrine and epinephrine neurons differentially affects circadian and stressor-stimulated corticosterone secretion. Endocrinology. 144: 1357-67. PMID 12639919 DOI: 10.1210/En.2002-221076 |
0.389 |
|
| 2003 |
Fraley GS, Ritter S. Immunolesion of norepinephrine and epinephrine afferents to medial hypothalamus alters basal and 2-deoxy-D-glucose-induced neuropeptide Y and agouti gene-related protein messenger ribonucleic acid expression in the arcuate nucleus. Endocrinology. 144: 75-83. PMID 12488332 DOI: 10.1210/En.2002-220659 |
0.485 |
|
| 2002 |
Fraley GS, Dinh TT, Ritter S. Immunotoxic catecholamine lesions attenuate 2DG-induced increase of AGRP mRNA. Peptides. 23: 1093-9. PMID 12126736 DOI: 10.1016/S0196-9781(02)00044-X |
0.425 |
|
| 2001 |
Sanders NM, Ritter S. Acute 2DG-induced glucoprivation or dexamethasone abolishes 2DG-induced glucoregulatory responses to subsequent glucoprivation. Diabetes. 50: 2831-6. PMID 11723067 |
0.318 |
|
| 2001 |
Ritter S, Bugarith K, Dinh TT. Immunotoxic destruction of distinct catecholamine subgroups produces selective impairment of glucoregulatory responses and neuronal activation. The Journal of Comparative Neurology. 432: 197-216. PMID 11241386 DOI: 10.1002/Cne.1097 |
0.798 |
|
| 2000 |
Sanders NM, Ritter S. Repeated 2-deoxy-D-glucose-induced glucoprivation attenuates Fos expression and glucoregulatory responses during subsequent glucoprivation. Diabetes. 49: 1865-74. PMID 11078453 |
0.383 |
|
| 2000 |
Ritter S, Dinh TT, Zhang Y. Localization of hindbrain glucoreceptive sites controlling food intake and blood glucose. Brain Research. 856: 37-47. PMID 10677609 DOI: 10.1016/S0006-8993(99)02327-6 |
0.426 |
|
| 1999 |
Wiater MF, Ritter S. Leptin does not attenuate the hyperphagia induced by 2-deoxy-D-glucose. Annals of the New York Academy of Sciences. 892: 334-6. PMID 10842678 DOI: 10.1111/J.1749-6632.1999.Tb07811.X |
0.762 |
|
| 1999 |
Ritter S, Ritter JB, Cromer L. 2-Deoxy-D-glucose and mercaptoacetate induce different patterns of macronutrient ingestion. Physiology & Behavior. 66: 709-15. PMID 10386918 DOI: 10.1016/S0031-9384(99)00009-8 |
0.461 |
|
| 1998 |
Ritter S, Llewellyn-Smith I, Dinh TT. Subgroups of hindbrain catecholamine neurons are selectively activated by 2-deoxy-D-glucose induced metabolic challenge. Brain Research. 805: 41-54. PMID 9733914 DOI: 10.1016/S0006-8993(98)00655-6 |
0.426 |
|
| 1998 |
Koegler FH, Ritter S. Galanin injection into the nucleus of the solitary tract stimulates feeding in rats with lesions of the paraventricular nucleus of the hypothalamus. Physiology & Behavior. 63: 521-7. PMID 9523894 DOI: 10.1016/S0031-9384(97)00480-0 |
0.803 |
|
| 1997 |
Koegler FH, Ritter S. Aqueduct occlusion does not impair feeding induced by either third or fourth ventricle galanin injection. Obesity Research. 5: 262-7. PMID 9192401 DOI: 10.1002/J.1550-8528.1997.Tb00301.X |
0.775 |
|
| 1996 |
Koegler FH, Ritter S. Feeding induced by pharmacological blockade of fatty acid metabolism is selectively attenuated by hindbrain injections of the galanin receptor antagonist, M40. Obesity Research. 4: 329-36. PMID 8822757 DOI: 10.1002/J.1550-8528.1996.Tb00240.X |
0.806 |
|
| 1996 |
Singer-Koegler LK, Magluyan P, Ritter S. The effects of low-, medium-, and high-fat diets on 2-deoxy-D-glucose- and mercaptoacetate-induced feeding. Physiology & Behavior. 60: 321-3. PMID 8804684 DOI: 10.1016/0031-9384(95)02142-6 |
0.436 |
|
| 1996 |
Singer LK, Ritter S. Intraventricular glucose blocks feeding induced by 2-deoxy-D-glucose but not mercaptoacetate. Physiology & Behavior. 59: 921-3. PMID 8778887 DOI: 10.1016/0031-9384(95)02144-2 |
0.418 |
|
| 1995 |
Ritter S, Scheurink A, Singer LK. 2-Deoxy-D-glucose but not 2-mercaptoacetate increases Fos-like immunoreactivity in adrenal medulla and sympathetic preganglionic neurons. Obesity Research. 3: 729S-734S. PMID 8653555 DOI: 10.1002/J.1550-8528.1995.Tb00492.X |
0.453 |
|
| 1995 |
Ritter S, Hutton B. Mercaptoacetate-induced feeding is impaired by central nucleus of the amygdala lesions. Physiology & Behavior. 58: 1215-20. PMID 8623023 DOI: 10.1016/0031-9384(95)02050-0 |
0.506 |
|
| 1995 |
van Dijk G, Scheurink A, Ritter S, Steffens A. Glucose homeostasis and sympathoadrenal activity in mercaptoacetate-treated rats. Physiology & Behavior. 57: 759-64. PMID 7777614 DOI: 10.1016/0031-9384(94)00323-8 |
0.398 |
|
| 1994 |
Singer LK, Ritter S. Differential effects of infused nutrients on 2-deoxy-D-glucose- and 2-mercaptoacetate-induced feeding. Physiology & Behavior. 56: 193-6. PMID 8084901 DOI: 10.1016/0031-9384(94)90279-8 |
0.475 |
|
| 1994 |
Ritter S, Dinh TT, Friedman MI. Induction of Fos-like immunoreactivity (Fos-li) and stimulation of feeding by 2,5-anhydro-D-mannitol (2,5-AM) require the vagus nerve. Brain Research. 646: 53-64. PMID 8055341 DOI: 10.1016/0006-8993(94)90057-4 |
0.378 |
|
| 1994 |
Ritter S, Dinh TT. 2-Mercaptoacetate and 2-deoxy-D-glucose induce Fos-like immunoreactivity in rat brain. Brain Research. 641: 111-20. PMID 8019836 DOI: 10.1016/0006-8993(94)91822-8 |
0.462 |
|
| 1994 |
Ritter S. Multiple metabolic controls of feeding. Appetite. 23: 199. PMID 7864618 DOI: 10.1006/Appe.1994.1052 |
0.326 |
|
| 1994 |
Ritter S, Singer L, Koegler F. Neural pathways for control of feeding by central and peripheral metabolic fuels Pathophysiology. 1: 89. DOI: 10.1016/0928-4680(94)90203-8 |
0.745 |
|
| 1993 |
Scheurink A, Ritter S. Sympathoadrenal responses to glucoprivation and lipoprivation in rats. Physiology & Behavior. 53: 995-1000. PMID 8511217 DOI: 10.1016/0031-9384(93)90279-O |
0.383 |
|
| 1993 |
Calingasan NY, Ritter S. Lateral parabrachial subnucleus lesions abolish feeding induced by mercaptoacetate but not by 2-deoxy-D-glucose. The American Journal of Physiology. 265: R1168-78. PMID 8238620 DOI: 10.1152/Ajpregu.1993.265.5.R1168 |
0.476 |
|
| 1992 |
Ritter S, Dinh TT. Age-related changes in capsaicin-induced degeneration in rat brain. The Journal of Comparative Neurology. 318: 103-16. PMID 1583153 DOI: 10.1002/Cne.903180108 |
0.386 |
|
| 1992 |
Calingasan NY, Ritter S. Hypothalamic paraventricular nucleus lesions do not abolish glucoprivic or lipoprivic feeding. Brain Research. 595: 25-31. PMID 1467956 DOI: 10.1016/0006-8993(92)91448-N |
0.505 |
|
| 1992 |
Calingasan N, Ritter S, Ritter R, Brenner L. Low-dose near-celiac arterial cholecystokinin suppresses food intake in rats. The American Journal of Physiology. 263: R572-7. PMID 1415643 DOI: 10.1152/Ajpregu.1992.263.3.R572 |
0.409 |
|
| 1992 |
Calingasan NY, Ritter S. Presence of galanin in rat vagal sensory neurons: evidence from immunohistochemistry and in situ hybridization. Journal of the Autonomic Nervous System. 40: 229-38. PMID 1281181 DOI: 10.1016/0165-1838(92)90205-U |
0.405 |
|
| 1992 |
Ritter S, Dinh TT. Capsaicin: A Probe for Studying Specific Neuronal Populations in Brain and Retina Methods in Neurosciences. 8: 118-136. DOI: 10.1016/B978-0-12-185266-5.50014-2 |
0.366 |
|
| 1992 |
Scheurink A, Ritter S, Dijk vG, Steffens A. Glucose homeostasis and sympathoadrenal activity after glucoprivation and lipoprivation in rats Appetite. 19: 215-215. DOI: 10.1016/0195-6663(92)90135-S |
0.365 |
|
| 1991 |
Ritter S, Dinh TT. Prior optic nerve transection reduces capsaicin-induced degeneration in rat subcortical visual structures. The Journal of Comparative Neurology. 308: 79-90. PMID 1714924 DOI: 10.1002/Cne.903080108 |
0.338 |
|
| 1990 |
Ritter S, Taylor JS. Vagal sensory neurons are required for lipoprivic but not glucoprivic feeding in rats. The American Journal of Physiology. 258: R1395-401. PMID 2360688 DOI: 10.1152/Ajpregu.1990.258.6.R1395 |
0.501 |
|
| 1990 |
Ritter S, Dinh TT. Capsaicin-induced neuronal degeneration in the brain and retina of preweanling rats. The Journal of Comparative Neurology. 296: 447-61. PMID 2358547 DOI: 10.1002/Cne.902960310 |
0.408 |
|
| 1990 |
Ritter S, Ritter RC. Capsaicin-sensitivity and the sensory vagus: Do these exceptions prove or disprove the B-neuron rule for autonomic afferents? Behavioral and Brain Sciences. 13: 315-316. DOI: 10.1017/S0140525X00078912 |
0.315 |
|
| 1989 |
Ritter S, Taylor JS. Capsaicin abolishes lipoprivic but not glucoprivic feeding in rats. The American Journal of Physiology. 256: R1232-9. PMID 2735449 DOI: 10.1152/Ajpregu.1989.256.6.R1232 |
0.509 |
|
| 1989 |
Ritter RC, Ritter S, Ewart WR, Wingate DL. Capsaicin attenuates hindbrain neuron responses to circulating cholecystokinin. The American Journal of Physiology. 257: R1162-8. PMID 2589542 DOI: 10.1152/Ajpregu.1989.257.5.R1162 |
0.385 |
|
| 1989 |
Weatherford SC, Ritter S. Doses of cholecystokinin and glucagon that combine to inhibit feeding do not affect gastric emptying in the rat Annals of the New York Academy of Sciences. 575: 592-595. DOI: 10.1111/J.1749-6632.1989.Tb53306.X |
0.304 |
|
| 1988 |
Weatherford SC, Ritter S. Lesion of vagal afferent terminals impairs glucagon-induced suppression of food intake Physiology and Behavior. 43: 645-650. PMID 3200921 DOI: 10.1016/0031-9384(88)90220-X |
0.436 |
|
| 1988 |
Ritter S, Dinh TT. Capsaicin-induced neuronal degeneration: Silver impregnation of cell bodies, axons, and terminals in the central nervous system of the adult rat Journal of Comparative Neurology. 271: 79-90. PMID 2454961 DOI: 10.1002/Cne.902710109 |
0.405 |
|
| 1987 |
Ritter S, Stone SL. Area postrema lesions block feeding induced by systemic injections of monosodium glutamate Physiology and Behavior. 41: 21-24. PMID 3685149 DOI: 10.1016/0031-9384(87)90125-9 |
0.433 |
|
| 1986 |
Ritter S, Weatherford SC, Stone SL. Glucagon-induced inhibition of feeding is impaired by hepatic portal alloxan injection American Journal of Physiology - Regulatory Integrative and Comparative Physiology. 250: 19/4. PMID 3963237 DOI: 10.1152/Ajpregu.1986.250.4.R682 |
0.461 |
|
| 1986 |
Reddy VM, Meharg SS, Ritter S. Dose-related stimulation of feeding by systemic injections of monosodium glutamate Physiology and Behavior. 38: 465-469. PMID 3823160 DOI: 10.1016/0031-9384(86)90412-9 |
0.427 |
|
| 1986 |
Weatherford SC, Ritter S. Glucagon satiety: Diurnal variation after hepatic branch vagotomy or intraportal alloxan Brain Research Bulletin. 17: 545-549. PMID 3779455 DOI: 10.1016/0361-9230(86)90224-8 |
0.39 |
|
| 1986 |
Weick BG, Ritter S. Stimulation of insulin release and suppression of feeding by hepatic portal glucagon infusion in rats Physiology and Behavior. 38: 531-536. PMID 3547424 DOI: 10.1016/0031-9384(86)90421-X |
0.383 |
|
| 1985 |
Murnane JM, Ritter S. Alloxan-induced glucoprivic feeding deficits are blocked by D-glucose and amygdalin Pharmacology, Biochemistry and Behavior. 22: 407-413. PMID 3991757 DOI: 10.1016/0091-3057(85)90041-3 |
0.397 |
|
| 1985 |
Murnane JM, Ritter S. Intraventricular alloxan impairs feeding to both central and systemic glucoprivation Physiology and Behavior. 34: 609-613. PMID 2989961 DOI: 10.1016/0031-9384(85)90056-3 |
0.428 |
|
| 1983 |
Weick BG, Ritter S, McCarty R. Plasma catecholamines in fasted and sucrose supplemented rats Physiology and Behavior. 30: 247-252. PMID 6844438 DOI: 10.1016/0031-9384(83)90014-8 |
0.396 |
|
| 1982 |
Ritter S, Strang M. Fourth ventricular alloxan injection causes feeding but not hyperglycemia in rats Brain Research. 249: 198-201. PMID 7139297 DOI: 10.1016/0006-8993(82)90190-1 |
0.482 |
|
| 1982 |
Ritter S, Murnane JM, Ladenheim EE. Glucoprivic feeding is impaired by lateral or fourth ventricular alloxan injection. The American Journal of Physiology. 243: R312-7. PMID 6214195 DOI: 10.1152/Ajpregu.1982.243.3.R312 |
0.457 |
|
| 1981 |
Ritter S, Bellin SI, Pelzer NL. The role of gustatory and postingestive signals in the termination of delayed glucoprivic feeding and hypothalamic norepinephrine turnover Journal of Neuroscience. 1: 1354-1360. PMID 7320750 DOI: 10.1523/Jneurosci.01-12-01354.1981 |
0.526 |
|
| 1981 |
Bellin SI, Ritter S. Disparate effects of infused nutrients on delayed glucoprivic feeding and hypothalamic norepinephrine turnover Journal of Neuroscience. 1: 1347-1353. PMID 7033477 |
0.368 |
|
| 1981 |
Bellin SI, Ritter S. Insulin-induced elevation of hypothalamic norepinephrine turnover persists after glucorestoration unless feeding occurs Brain Research. 217: 327-337. PMID 7018642 DOI: 10.1016/0006-8993(81)90008-1 |
0.454 |
|
| 1980 |
Ritter S, McGlone JJ, Kelley KW. Absence of lithium-induced taste aversion after area postrema lesion Brain Research. 201: 501-506. PMID 7417860 DOI: 10.1016/0006-8993(80)91061-6 |
0.337 |
|
| 1980 |
McGlone JJ, Ritter S, Kelley KW. The antiaggressive effect of lithium is abolished by area postrema lesion Physiology and Behavior. 24: 1095-1100. PMID 7191119 DOI: 10.1016/0031-9384(80)90053-0 |
0.326 |
|
| 1978 |
Ritter S, Pelzer NL, Ritter RC. Absence of glucoprivic feeding after stress suggest impairment of noradrenergic neuron function. Brain Research. 149: 399-411. PMID 566607 DOI: 10.1016/0006-8993(78)90483-3 |
0.352 |
|
| 1977 |
Ritter S, Ritter RC. Protection against stress-induced brain norepinephrine depletion after repeated 2-deoxy-D-glucose administration. Brain Research. 127: 179-84. PMID 861752 DOI: 10.1016/0006-8993(77)90391-2 |
0.326 |
|
| 1975 |
Belluzzi JD, Ritter S, Wise CD, Stein L. Substantia nigra self-stimulation: dependence on noradrenergic reward pathways. Behavioral Biology. 13: 103-11. PMID 1111501 DOI: 10.1016/S0091-6773(75)90853-6 |
0.302 |
|
| 1975 |
Ritter S, Wise CD, Stein L. Neurochemical regulation of feeding in the rat: Facilitation by a-noradrenergic, but not dopaminergic, receptor stimulants Journal of Comparative and Physiological Psychology. 88: 778-784. PMID 239031 DOI: 10.1037/H0076402 |
0.421 |
|
| 1974 |
Stein L, Belluzzi JD, Ritter S, Wise CD. Self-stimulation reward pathways: norepinephrine vs dopamine. Journal of Psychiatric Research. 11: 115-24. PMID 4461783 DOI: 10.1016/0022-3956(74)90082-X |
0.312 |
|
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