Frederik Wirtz-Peitz - Publications

Institute of Molecular Biotechnology, Wien, Wien, Austria 
Asymmetric cell division, Par proteins

14 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2020 Kim K, Lane EA, Saftien A, Wang H, Xu Y, Wirtz-Peitz F, Perrimon N. as a model for studying cystic fibrosis pathophysiology of the gastrointestinal system. Proceedings of the National Academy of Sciences of the United States of America. PMID 32345720 DOI: 10.1073/pnas.1913127117  0.8
2019 Silver JT, Wirtz-Peitz F, Simões S, Pellikka M, Yan D, Binari R, Nishimura T, Li Y, Harris TJC, Perrimon N, Tepass U. Apical polarity proteins recruit the RhoGEF Cysts to promote junctional myosin assembly. The Journal of Cell Biology. PMID 31409654 DOI: 10.1083/jcb.201807106  0.8
2017 Rajan A, Housden BE, Wirtz-Peitz F, Holderbaum L, Perrimon N. A Mechanism Coupling Systemic Energy Sensing to Adipokine Secretion. Developmental Cell. 43: 83-98.e6. PMID 29017032 DOI: 10.1016/j.devcel.2017.09.007  0.8
2015 Chen CL, Hu Y, Udeshi ND, Lau TY, Wirtz-Peitz F, He L, Ting AY, Carr SA, Perrimon N. Proteomic mapping in live Drosophila tissues using an engineered ascorbate peroxidase. Proceedings of the National Academy of Sciences of the United States of America. PMID 26362788 DOI: 10.1073/pnas.1515623112  0.32
2012 Neumüller RA, Wirtz-Peitz F, Lee S, Kwon Y, Buckner M, Hoskins RA, Venken KJ, Bellen HJ, Mohr SE, Perrimon N. Stringent analysis of gene function and protein-protein interactions using fluorescently tagged genes. Genetics. 190: 931-40. PMID 22174071 DOI: 10.1534/genetics.111.136465  0.32
2009 Sanders PG, Muñoz-Descalzo S, Balayo T, Wirtz-Peitz F, Hayward P, Arias AM. Ligand-independent traffic of Notch buffers activated Armadillo in Drosophila. Plos Biology. 7: e1000169. PMID 19668359 DOI: 10.1371/journal.pbio.1000169  0.32
2009 Wirtz-Peitz F, Zallen JA. Junctional trafficking and epithelial morphogenesis. Current Opinion in Genetics & Development. 19: 350-6. PMID 19559596 DOI: 10.1016/j.gde.2009.04.011  0.32
2008 Wirtz-Peitz F, Nishimura T, Knoblich JA. Linking cell cycle to asymmetric division: Aurora-A phosphorylates the Par complex to regulate Numb localization. Cell. 135: 161-73. PMID 18854163 DOI: 10.1016/j.cell.2008.07.049  0.32
2006 Hutterer A, Berdnik D, Wirtz-Peitz F, Zigman M, Schleiffer A, Knoblich JA. Mitotic activation of the kinase Aurora-A requires its binding partner Bora. Developmental Cell. 11: 147-57. PMID 16890155 DOI: 10.1016/j.devcel.2006.06.002  0.32
2006 Wirtz-Peitz F, Knoblich JA. Lethal giant larvae take on a life of their own. Trends in Cell Biology. 16: 234-41. PMID 16616850 DOI: 10.1016/j.tcb.2006.03.006  0.32
2006 Langdon T, Hayward P, Brennan K, Wirtz-Peitz F, Sanders P, Zecchini V, Friday A, Balayo T, Martinez Arias A. Notch receptor encodes two structurally separable functions in Drosophila: a genetic analysis. Developmental Dynamics : An Official Publication of the American Association of Anatomists. 235: 998-1013. PMID 16534797 DOI: 10.1002/dvdy.20735  0.32
2005 Mayer B, Emery G, Berdnik D, Wirtz-Peitz F, Knoblich JA. Quantitative analysis of protein dynamics during asymmetric cell division. Current Biology : Cb. 15: 1847-54. PMID 16243032 DOI: 10.1016/j.cub.2005.08.067  0.32
2005 Jafar-Nejad H, Andrews HK, Acar M, Bayat V, Wirtz-Peitz F, Mehta SQ, Knoblich JA, Bellen HJ. Sec15, a component of the exocyst, promotes notch signaling during the asymmetric division of Drosophila sensory organ precursors. Developmental Cell. 9: 351-63. PMID 16137928 DOI: 10.1016/j.devcel.2005.06.010  0.32
2005 Emery G, Hutterer A, Berdnik D, Mayer B, Wirtz-Peitz F, Gaitan MG, Knoblich JA. Asymmetric Rab 11 endosomes regulate delta recycling and specify cell fate in the Drosophila nervous system. Cell. 122: 763-73. PMID 16137758 DOI: 10.1016/j.cell.2005.08.017  0.32
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