Yi Guan, Ph.D. - Publications
Affiliations: | 2002 | University of California, Berkeley, Berkeley, CA, United States |
Area:
Cell Biology, Molecular Biology, OncologyYear | Citation | Score | |||
---|---|---|---|---|---|
2021 | Zhu S, Yang N, Guan Y, Wang X, Zang G, Lv X, Deng S, Wang W, Li T, Chen J. GDF15 promotes glioma stem cell-like phenotype via regulation of ERK1/2-c-Fos-LIF signaling. Cell Death Discovery. 7: 3. PMID 33431816 DOI: 10.1038/s41420-020-00395-8 | 0.327 | |||
2004 | Guan Y, Rubenstein NM, Failor KL, Woo PL, Firestone GL. Glucocorticoids control beta-catenin protein expression and localization through distinct pathways that can be uncoupled by disruption of signaling events required for tight junction formation in rat mammary epithelial tumor cells. Molecular Endocrinology (Baltimore, Md.). 18: 214-27. PMID 14551262 DOI: 10.1210/Me.2003-0014 | 0.514 | |||
2003 | Rubenstein NM, Guan Y, Woo PL, Firestone GL. Glucocorticoid down-regulation of RhoA is required for the steroid-induced organization of the junctional complex and tight junction formation in rat mammary epithelial tumor cells. The Journal of Biological Chemistry. 278: 10353-60. PMID 12525486 DOI: 10.1074/Jbc.M213121200 | 0.71 | |||
2002 | Guan Y, Woo PL, Rubenstein NM, Firestone GL. Transforming growth factor-alpha abrogates the glucocorticoid stimulation of tight junction formation and reverses the steroid-induced down-regulation of fascin in rat mammary epithelial tumor cells by a Ras-dependent pathway. Experimental Cell Research. 273: 1-11. PMID 11795941 DOI: 10.1006/Excr.2001.5415 | 0.71 | |||
1999 | Woo PL, Ching D, Guan Y, Firestone GL. Requirement for Ras and phosphatidylinositol 3-kinase signaling uncouples the glucocorticoid-induced junctional organization and transepithelial electrical resistance in mammary tumor cells. The Journal of Biological Chemistry. 274: 32818-28. PMID 10551843 DOI: 10.1074/Jbc.274.46.32818 | 0.666 | |||
Show low-probability matches. |