Year |
Citation |
Score |
2007 |
Denault JB, Eckelman BP, Shin H, Pop C, Salvesen GS. Caspase 3 attenuates XIAP (X-linked inhibitor of apoptosis protein)-mediated inhibition of caspase 9. The Biochemical Journal. 405: 11-9. PMID 17437405 DOI: 10.1042/Bj20070288 |
0.718 |
|
2005 |
Scott FL, Denault JB, Riedl SJ, Shin H, Renatus M, Salvesen GS. XIAP inhibits caspase-3 and -7 using two binding sites: evolutionarily conserved mechanism of IAPs. The Embo Journal. 24: 645-55. PMID 15650747 DOI: 10.1038/Sj.Emboj.7600544 |
0.775 |
|
2005 |
Vucic D, Franklin MC, Wallweber HJ, Das K, Eckelman BP, Shin H, Elliott LO, Kadkhodayan S, Deshayes K, Salvesen GS, Fairbrother WJ. Engineering ML-IAP to produce an extraordinarily potent caspase 9 inhibitor: implications for Smac-dependent anti-apoptotic activity of ML-IAP. The Biochemical Journal. 385: 11-20. PMID 15485396 DOI: 10.1042/Bj20041108 |
0.715 |
|
2005 |
Shin H, Renatus M, Eckelman BP, Nunes VA, Sampaio CA, Salvesen GS. The BIR domain of IAP-like protein 2 is conformationally unstable: implications for caspase inhibition. The Biochemical Journal. 385: 1-10. PMID 15485395 DOI: 10.1042/Bj20041107 |
0.713 |
|
2003 |
Kashkar H, Haefs C, Shin H, Hamilton-Dutoit SJ, Salvesen GS, Kronke M, Jurgensmeier JM. XIAP-mediated caspase inhibition in Hodgkin's lymphoma-derived B cells. The Journal of Experimental Medicine. 198: 341-7. PMID 12874265 DOI: 10.1084/Jem.20021279 |
0.681 |
|
2003 |
Shin H, Okada K, Wilkinson JC, Solomon KM, Duckett CS, Reed JC, Salvesen GS. Identification of ubiquitination sites on the X-linked inhibitor of apoptosis protein Biochemical Journal. 373: 965-971. PMID 12747801 DOI: 10.1042/Bj20030583 |
0.692 |
|
2003 |
Boatright KM, Renatus M, Scott FL, Sperandio S, Shin H, Pedersen IM, Ricci JE, Edris WA, Sutherlin DP, Green DR, Salvesen GS. A unified model for apical caspase activation. Molecular Cell. 11: 529-41. PMID 12620239 DOI: 10.1016/S1097-2765(03)00051-0 |
0.718 |
|
2001 |
Forcet C, Ye X, Granger L, Corset V, Shin H, Bredesen DE, Mehlen P. The dependence receptor DCC (deleted in colorectal cancer) defines an alternative mechanism for caspase activation Proceedings of the National Academy of Sciences of the United States of America. 98: 3416-3421. PMID 11248093 DOI: 10.1073/Pnas.051378298 |
0.557 |
|
1999 |
Ye X, Mehlen P, Rabizadeh S, VanArsdale T, Zhang H, Shin H, Wang JJL, Leo E, Zapata J, Hauser CA, Reed JC, Bredesen DE. TRAF Family Proteins Interact with the Common Neurotrophin Receptor and Modulate Apoptosis Induction Journal of Biological Chemistry. 274: 30202-30208. PMID 10514511 DOI: 10.1074/Jbc.274.42.30202 |
0.51 |
|
1998 |
Stennicke HR, Jürgensmeier JM, Shin H, Deveraux Q, Wolf BB, Yang X, Zhou Q, Ellerby HM, Ellerby LM, Bredesen D, Green DR, Reed JC, Froelich CJ, Salvesen GS. Pro-caspase-3 is a major physiologic target of caspase-8. The Journal of Biological Chemistry. 273: 27084-90. PMID 9765224 DOI: 10.1074/Jbc.273.42.27084 |
0.725 |
|
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