Year |
Citation |
Score |
2013 |
Kobayashi SD, Olsen RJ, LaCasse RA, Safronetz D, Ashraf M, Porter AR, Braughton KR, Feldmann F, Clifton DR, Kash JC, Bailey JR, Gardner DJ, Otto M, Brining DL, Kreiswirth BN, et al. Seasonal H3N2 influenza A virus fails to enhance Staphylococcus aureus co-infection in a non-human primate respiratory tract infection model. Virulence. 4: 707-15. PMID 24104465 DOI: 10.4161/Viru.26572 |
0.399 |
|
2013 |
Patton TG, Brandt KS, Nolder C, Clifton DR, Carroll JA, Gilmore RD. Borrelia burgdorferi bba66 gene inactivation results in attenuated mouse infection by tick transmission. Infection and Immunity. 81: 2488-98. PMID 23630963 DOI: 10.1128/Iai.00140-13 |
0.523 |
|
2010 |
Gilmore RD, Howison RR, Dietrich G, Patton TG, Clifton DR, Carroll JA. The bba64 gene of Borrelia burgdorferi, the Lyme disease agent, is critical for mammalian infection via tick bite transmission. Proceedings of the National Academy of Sciences of the United States of America. 107: 7515-20. PMID 20368453 DOI: 10.1073/Pnas.1000268107 |
0.526 |
|
2008 |
Hughes JL, Nolder CL, Nowalk AJ, Clifton DR, Howison RR, Schmit VL, Gilmore RD, Carroll JA. Borrelia burgdorferi surface-localized proteins expressed during persistent murine infection are conserved among diverse Borrelia spp. Infection and Immunity. 76: 2498-511. PMID 18390998 DOI: 10.1128/Iai.01583-07 |
0.488 |
|
2007 |
Gilmore RD, Howison RR, Schmit VL, Nowalk AJ, Clifton DR, Nolder C, Hughes JL, Carroll JA. Temporal expression analysis of the Borrelia burgdorferi paralogous gene family 54 genes BBA64, BBA65, and BBA66 during persistent infection in mice. Infection and Immunity. 75: 2753-64. PMID 17371862 DOI: 10.1128/Iai.00037-07 |
0.511 |
|
2006 |
Clifton DR, Nolder CL, Hughes JL, Nowalk AJ, Carroll JA. Regulation and expression of bba66 encoding an immunogenic infection-associated lipoprotein in Borrelia burgdorferi. Molecular Microbiology. 61: 243-58. PMID 16824109 DOI: 10.1111/J.1365-2958.2006.05224.X |
0.468 |
|
2006 |
Stewart PE, Wang X, Bueschel DM, Clifton DR, Grimm D, Tilly K, Carroll JA, Weis JJ, Rosa PA. Delineating the requirement for the Borrelia burgdorferi virulence factor OspC in the mammalian host. Infection and Immunity. 74: 3547-53. PMID 16714587 DOI: 10.1128/Iai.00158-06 |
0.53 |
|
2005 |
Clifton DR, Rydkina E, Huyck H, Pryhuber G, Freeman RS, Silverman DJ, Sahni SK. Expression and secretion of chemotactic cytokines IL-8 and MCP-1 by human endothelial cells after Rickettsia rickettsii infection: regulation by nuclear transcription factor NF-kappaB. International Journal of Medical Microbiology : Ijmm. 295: 267-78. PMID 16128401 DOI: 10.1016/J.Ijmm.2005.05.006 |
0.48 |
|
2005 |
Clifton DR, Rydkina E, Freeman RS, Sahni SK. NF-kappaB activation during Rickettsia rickettsii infection of endothelial cells involves the activation of catalytic IkappaB kinases IKKalpha and IKKbeta and phosphorylation-proteolysis of the inhibitor protein IkappaBalpha. Infection and Immunity. 73: 155-65. PMID 15618150 DOI: 10.1128/Iai.73.1.155-165.2005 |
0.567 |
|
2004 |
Clifton DR, Fields KA, Grieshaber SS, Dooley CA, Fischer ER, Mead DJ, Carabeo RA, Hackstadt T. A chlamydial type III translocated protein is tyrosine-phosphorylated at the site of entry and associated with recruitment of actin. Proceedings of the National Academy of Sciences of the United States of America. 101: 10166-71. PMID 15199184 DOI: 10.1073/Pnas.0402829101 |
0.303 |
|
1998 |
Clifton DR, Goss RA, Sahni SK, van Antwerp D, Baggs RB, Marder VJ, Silverman DJ, Sporn LA. NF-kappa B-dependent inhibition of apoptosis is essential for host cellsurvival during Rickettsia rickettsii infection. Proceedings of the National Academy of Sciences of the United States of America. 95: 4646-51. PMID 9539792 DOI: 10.1073/Pnas.95.8.4646 |
0.426 |
|
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