| Year |
Citation |
Score |
| 2024 |
Hernandez-Ono A, Zhao YP, Murray JW, Östlund C, Lee MJ, Shi A, Dauer WT, Worman HJ, Ginsberg HN, Shin JY. Dynamic regulation of hepatic lipid metabolism by torsinA and its activators. Jci Insight. PMID 38194265 DOI: 10.1172/jci.insight.175328 |
0.487 |
|
| 2023 |
Hernandez-Ono A, Zhao YP, Murray JW, Östlund C, Lee MJ, Shi A, Dauer WT, Worman HJ, Ginsberg HN, Shin JY. Functional interaction of torsinA and its activators in liver lipid metabolism. Biorxiv : the Preprint Server For Biology. PMID 37547008 DOI: 10.1101/2023.06.21.545957 |
0.503 |
|
| 2022 |
Östlund C, Hernandez-Ono A, Turk SJ, Dauer WT, Ginsberg HN, Worman HJ, Shin JY. Hepatocytes deficient in nuclear envelope protein lamina-associated polypeptide 1 are an ideal mammalian system to study intranuclear lipid droplets. Journal of Lipid Research. 100277. PMID 36100089 DOI: 10.1016/j.jlr.2022.100277 |
0.541 |
|
| 2022 |
Wang Y, Shilagardi K, Hsu T, Odinammadu KO, Maruyama T, Wu W, Lin CS, Damoci CB, Spear ED, Shin JY, Hsu W, Michaelis S, Worman HJ. Abolishing the prelamin A ZMPSTE24 cleavage site leads to progeroid phenotypes with near-normal longevity in mice. Proceedings of the National Academy of Sciences of the United States of America. 119. PMID 35197292 DOI: 10.1073/pnas.2118695119 |
0.528 |
|
| 2021 |
Shin JY, Worman HJ. Molecular Pathology of Laminopathies. Annual Review of Pathology. PMID 34672689 DOI: 10.1146/annurev-pathol-042220-034240 |
0.549 |
|
| 2021 |
Kronenberg-Tenga R, Tatli M, Eibauer M, Wu W, Shin JY, Bonne G, Worman HJ, Medalia O. A lamin A/C variant causing striated muscle disease provides insights into filament organization. Journal of Cell Science. PMID 33536248 DOI: 10.1242/jcs.256156 |
0.54 |
|
| 2019 |
Shin JY, Hernandez-Ono A, Fedotova T, Östlund C, Lee MJ, Gibeley SB, Liang CC, Dauer WT, Ginsberg HN, Worman HJ. Nuclear envelope-localized torsinA-LAP1 complex regulates hepatic VLDL secretion and steatosis. The Journal of Clinical Investigation. 130. PMID 31408437 DOI: 10.1172/Jci129769 |
0.518 |
|
| 2019 |
Wang Y, Shin JY, Nakanishi K, Homma S, Kim GJ, Tanji K, Joseph LC, Morrow JP, Stewart CL, Dauer WT, Worman HJ. Postnatal Development of Mice with Combined Genetic Depletions of Lamin A/C, Emerin and Lamina-associated Polypeptide 1. Human Molecular Genetics. PMID 31009944 DOI: 10.1093/Hmg/Ddz082 |
0.536 |
|
| 2016 |
Wu W, Chordia MD, Hart BP, Kumarasinghe ES, Ji MK, Bhargava A, Lawlor MW, Shin JY, Sera F, Homma S, Muchir A, Khire UR, Worman HJ. Macrocyclic MEK1/2 inhibitor with efficacy in a mouse model of cardiomyopathy caused by lamin A/C gene mutation. Bioorganic & Medicinal Chemistry. PMID 28011205 DOI: 10.1016/J.Bmc.2016.12.014 |
0.657 |
|
| 2016 |
Shin JY, Méndez-López I, Hong M, Wang Y, Tanji K, Wu W, Shugol L, Krauss RS, Dauer WT, Worman HJ. Lamina-associated polypeptide 1 is dispensable for embryonic myogenesis but required for postnatal skeletal muscle growth. Human Molecular Genetics. PMID 27798115 DOI: 10.1093/Hmg/Ddw368 |
0.521 |
|
| 2016 |
Wang Y, Lichter-Konecki U, Anyane-Yeboa K, Shaw JE, Lu JT, Östlund C, Shin JY, Clark LN, Gundersen GG, Nagy PL, Worman HJ. Mutation abolishing the ZMPSTE24 cleavage site in prelamin A causes a progeroid disorder. Journal of Cell Science. PMID 27034136 DOI: 10.1242/Jcs.187302 |
0.51 |
|
| 2014 |
Shin JY, Le Dour C, Sera F, Iwata S, Homma S, Joseph LC, Morrow JP, Dauer WT, Worman HJ. Depletion of lamina-associated polypeptide 1 from cardiomyocytes causes cardiac dysfunction in mice. Nucleus (Austin, Tex.). 5: 260-459. PMID 24859316 DOI: 10.4161/Nucl.29227 |
0.536 |
|
| 2014 |
Shin JY, Dauer WT, Worman HJ. Lamina-associated polypeptide 1: protein interactions and tissue-selective functions. Seminars in Cell & Developmental Biology. 29: 164-8. PMID 24508913 DOI: 10.1016/J.Semcdb.2014.01.010 |
0.567 |
|
| 2013 |
Shin JY, Méndez-López I, Wang Y, Hays AP, Tanji K, Lefkowitch JH, Schulze PC, Worman HJ, Dauer WT. Lamina-associated polypeptide-1 interacts with the muscular dystrophy protein emerin and is essential for skeletal muscle maintenance. Developmental Cell. 26: 591-603. PMID 24055652 DOI: 10.1016/J.Devcel.2013.08.012 |
0.546 |
|
| 2010 |
Bradford J, Shin JY, Roberts M, Wang CE, Sheng G, Li S, Li XJ. Mutant huntingtin in glial cells exacerbates neurological symptoms of Huntington disease mice. The Journal of Biological Chemistry. 285: 10653-61. PMID 20145253 DOI: 10.1074/jbc.M109.083287 |
0.553 |
|
| 2009 |
Bradford J, Shin JY, Roberts M, Wang CE, Li XJ, Li S. Expression of mutant huntingtin in mouse brain astrocytes causes age-dependent neurological symptoms. Proceedings of the National Academy of Sciences of the United States of America. 106: 22480-5. PMID 20018729 DOI: 10.1073/pnas.0911503106 |
0.561 |
|
| 2006 |
Cornett J, Smith L, Friedman M, Shin JY, Li XJ, Li SH. Context-dependent dysregulation of transcription by mutant huntingtin. The Journal of Biological Chemistry. 281: 36198-204. PMID 17012230 DOI: 10.1074/Jbc.M607839200 |
0.66 |
|
| 2006 |
Rong J, McGuire JR, Fang ZH, Sheng G, Shin JY, Li SH, Li XJ. Regulation of intracellular trafficking of huntingtin-associated protein-1 is critical for TrkA protein levels and neurite outgrowth. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 26: 6019-30. PMID 16738245 DOI: 10.1523/Jneurosci.1251-06.2006 |
0.69 |
|
| 2006 |
Shin J, Fang Z, Yu Z, Wang C, Li S, Li X. Correction: Expression of mutant huntingtin in glial cells contributes to neuronal excitotoxicity Journal of Cell Biology. 172: 953-953. DOI: 10.1083/Jcb.20050807220060228C |
0.532 |
|
| 2005 |
Shin JY, Fang ZH, Yu ZX, Wang CE, Li SH, Li XJ. Expression of mutant huntingtin in glial cells contributes to neuronal excitotoxicity. The Journal of Cell Biology. 171: 1001-12. PMID 16365166 DOI: 10.1083/Jcb.200508072 |
0.552 |
|
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