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Michael Gleimer, Ph.D. - Publications

Affiliations: 
2008 Stanford University, Palo Alto, CA 
Area:
Molecular Biology
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11 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2015 Goyos A, Guethlein LA, Horowitz A, Hilton HG, Gleimer M, Brodsky FM, Parham P. A Distinctive Cytoplasmic Tail Contributes to Low Surface Expression and Intracellular Retention of the Patr-AL MHC Class I Molecule. Journal of Immunology (Baltimore, Md. : 1950). 195: 3725-36. PMID 26371256 DOI: 10.4049/Jimmunol.1500397  0.581
2012 Gleimer M, von Boehmer H, Kreslavsky T. PLZF Controls the Expression of a Limited Number of Genes Essential for NKT Cell Function. Frontiers in Immunology. 3: 374. PMID 23267359 DOI: 10.3389/fimmu.2012.00374  0.315
2011 Gleimer M, Wahl AR, Hickman HD, Abi-Rached L, Norman PJ, Guethlein LA, Hammond JA, Draghi M, Adams EJ, Juo S, Jalili R, Gharizadeh B, Ronaghi M, Garcia KC, Hildebrand WH, et al. Although divergent in residues of the peptide binding site, conserved chimpanzee Patr-AL and polymorphic human HLA-A*02 have overlapping peptide-binding repertoires. Journal of Immunology (Baltimore, Md. : 1950). 186: 1575-88. PMID 21209280 DOI: 10.4049/Jimmunol.1002990  0.575
2009 Graef T, Moesta AK, Norman PJ, Abi-Rached L, Vago L, Older Aguilar AM, Gleimer M, Hammond JA, Guethlein LA, Bushnell DA, Robinson PJ, Parham P. KIR2DS4 is a product of gene conversion with KIR3DL2 that introduced specificity for HLA-A*11 while diminishing avidity for HLA-C. The Journal of Experimental Medicine. 206: 2557-72. PMID 19858347 DOI: 10.1084/Jem.20091010  0.472
2008 Moesta AK, Norman PJ, Yawata M, Yawata N, Gleimer M, Parham P. Synergistic polymorphism at two positions distal to the ligand-binding site makes KIR2DL2 a stronger receptor for HLA-C than KIR2DL3. Journal of Immunology (Baltimore, Md. : 1950). 180: 3969-79. PMID 18322206 DOI: 10.4049/Jimmunol.180.6.3969  0.698
2007 Norman PJ, Abi-Rached L, Gendzekhadze K, Korbel D, Gleimer M, Rowley D, Bruno D, Carrington CV, Chandanayingyong D, Chang YH, Crespí C, Saruhan-Direskeneli G, Fraser PA, Hameed K, Kamkamidze G, et al. Unusual selection on the KIR3DL1/S1 natural killer cell receptor in Africans. Nature Genetics. 39: 1092-9. PMID 17694054 DOI: 10.1038/Ng2111  0.691
2005 Draghi M, Yawata N, Gleimer M, Yawata M, Valiante NM, Parham P. Single-cell analysis of the human NK cell response to missing self and its inhibition by HLA class I. Blood. 105: 2028-35. PMID 15528315 DOI: 10.1182/Blood-2004-08-3174  0.631
2003 Pando MJ, Gardiner CM, Gleimer M, McQueen KL, Parham P. The protein made from a common allele of KIR3DL1 (3DL1*004) is poorly expressed at cell surfaces due to substitution at positions 86 in Ig domain 0 and 182 in Ig domain 1. Journal of Immunology (Baltimore, Md. : 1950). 171: 6640-9. PMID 14662867 DOI: 10.4049/Jimmunol.171.12.6640  0.557
2003 Gleimer M, Parham P. Stress management: MHC class I and class I-like molecules as reporters of cellular stress. Immunity. 19: 469-77. PMID 14563312 DOI: 10.1016/S1074-7613(03)00272-3  0.505
2003 Huppa JB, Gleimer M, Sumen C, Davis MM. Continuous T cell receptor signaling required for synapse maintenance and full effector potential. Nature Immunology. 4: 749-55. PMID 12858171 DOI: 10.1038/Ni951  0.314
2002 Lu LS, Tung J, Baumgarth N, Herman O, Gleimer M, Herzenberg LA, Herzenberg LA. Identification of a germ-line pro-B cell subset that distinguishes the fetal/neonatal from the adult B cell development pathway. Proceedings of the National Academy of Sciences of the United States of America. 99: 3007-12. PMID 11867763 DOI: 10.1073/Pnas.052715399  0.344
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