| Year |
Citation |
Score |
| 2023 |
Wang J, Joseph S, Vingill S, Dere E, Tatenhorst L, Ronnenberg A, Lingor P, Preisinger C, Ehrenreich H, Schulz JB, Stegmüller J. Loss of the parkinsonism-associated protein FBXO7 in glutamatergic forebrain neurons in mice leads to abnormal motor behavior and synaptic defects. Journal of Neurochemistry. PMID 37753846 DOI: 10.1111/jnc.15962 |
0.485 |
|
| 2021 |
Wang Q, Crnković V, Preisinger C, Stegmüller J. The parkinsonism-associated protein FBXO7 cooperates with the BAG6 complex in proteasome function and controls the subcellular localization of the complex. The Biochemical Journal. PMID 34060591 DOI: 10.1042/BCJ20201000 |
0.351 |
|
| 2021 |
Kawabe H, Stegmüller J. The role of E3 ubiquitin ligases in synapse function in the healthy and diseased brain. Molecular and Cellular Neurosciences. 112: 103602. PMID 33581237 DOI: 10.1016/j.mcn.2021.103602 |
0.365 |
|
| 2020 |
Stegmüller J. Restless in the mouse cage-A new genetic model for restless legs syndrome: An Editorial Highlight for "Deficiency of Meis1, a transcriptional regulator, in mice and worms:Neurochemical and behavioral characterizations with implications in the restless legs syndrome" on https://doi.org/10.1111/jnc.15177. Journal of Neurochemistry. PMID 33011998 DOI: 10.1111/jnc.15165 |
0.305 |
|
| 2016 |
Vingill S, Brockelt D, Lancelin C, Tatenhorst L, Dontcheva G, Preisinger C, Schwedhelm-Domeyer N, Joseph S, Mitkovski M, Goebbels S, Nave KA, Schulz JB, Marquardt T, Lingor P, Stegmüller J. Loss of FBXO7 (PARK15) results in reduced proteasome activity and models a parkinsonism-like phenotype in mice. The Embo Journal. PMID 27497298 DOI: 10.15252/Embj.201593585 |
0.466 |
|
| 2015 |
Mukherjee C, Holubowska A, Schwedhelm-Domeyer N, Mitkovski M, Lee SJ, Kannan M, Matz A, Vadhvani M, Stegmüller J. Loss of the neuron-specific F-box protein FBXO41 models an ataxia-like phenotype in mice with neuronal migration defects and degeneration in the cerebellum. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 35: 8701-17. PMID 26063905 DOI: 10.1523/Jneurosci.2133-14.2015 |
0.655 |
|
| 2015 |
Matz A, Lee SJ, Schwedhelm-Domeyer N, Zanini D, Holubowska A, Kannan M, Farnworth M, Jahn O, Göpfert MC, Stegmüller J. Regulation of neuronal survival and morphology by the E3 ubiquitin ligase RNF157. Cell Death and Differentiation. 22: 626-42. PMID 25342469 DOI: 10.1038/Cdd.2014.163 |
0.642 |
|
| 2013 |
Stegmüller J. The brake within: Mechanisms of intrinsic regulation of axon growth featuring the Cdh1-APC pathway Translational Neuroscience. 4. DOI: 10.2478/S13380-013-0125-2 |
0.347 |
|
| 2010 |
Stegmüller J, Bonni A. Destroy to create: E3 ubiquitin ligases in neurogenesis. F1000 Biology Reports. 2. PMID 20948796 DOI: 10.3410/B2-38 |
0.535 |
|
| 2008 |
Oberdoerffer P, Michan S, McVay M, Mostoslavsky R, Vann J, Park SK, Hartlerode A, Stegmuller J, Hafner A, Loerch P, Wright SM, Mills KD, Bonni A, Yankner BA, Scully R, et al. SIRT1 redistribution on chromatin promotes genomic stability but alters gene expression during aging. Cell. 135: 907-18. PMID 19041753 DOI: 10.1016/J.Cell.2008.10.025 |
0.5 |
|
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