| Year |
Citation |
Score |
| 2023 |
Cain SM, Alles SRA, Gopaul R, Bernier LP, Yung AC, Bauman A, Yang Y, Baker GB, Kozlowski P, MacVicar BA, Snutch TP. Chronic pregabalin treatment protects against spreading depolarization and alters hippocampal synaptic characteristics in a model of familial hemiplegic migraine-type 1. Molecular Brain. 16: 76. PMID 37924146 DOI: 10.1186/s13041-023-01062-6 |
0.351 |
|
| 2022 |
Cain SM, Bernier LP, Zhang Y, Yung AC, Kass J, Bohnet B, Yang Y, Gopaul R, Kozlowski P, MacVicar BA, Snutch TP. Hyperexcitable superior colliculus and fatal brainstem spreading depolarization in a model of Sudden Unexpected Death in Epilepsy. Brain Communications. 4: fcac006. PMID 35474853 DOI: 10.1093/braincomms/fcac006 |
0.322 |
|
| 2022 |
McElroy DL, Roebuck AJ, Greba Q, Garai S, Brandt AL, Yilmaz O, Cain SM, Snutch TP, Thakur GA, Laprairie RB, Howland JG. The type 1 cannabinoid receptor positive allosteric modulators GAT591 and GAT593 reduce spike-and-wave discharges in Genetic Absence Epilepsy Rats from Strasbourg. Ibro Neuroscience Reports. 12: 121-130. PMID 35128516 DOI: 10.1016/j.ibneur.2022.01.006 |
0.317 |
|
| 2021 |
Roebuck AJ, Greba Q, Smolyakova AM, Alaverdashvili M, Marks WN, Garai S, Baglot SL, Petrie G, Cain SM, Snutch TP, Thakur GA, Hill MN, Howland JG, Laprairie RB. Positive allosteric modulation of type 1 cannabinoid receptors reduces spike-and-wave discharges in Genetic Absence Epilepsy Rats from Strasbourg. Neuropharmacology. 108553. PMID 33845076 DOI: 10.1016/j.neuropharm.2021.108553 |
0.308 |
|
| 2020 |
Alles SRA, Cain SM, Snutch TP. Pregabalin as a Pain Therapeutic: Beyond Calcium Channels. Frontiers in Cellular Neuroscience. 14: 83. PMID 32351366 DOI: 10.3389/Fncel.2020.00083 |
0.445 |
|
| 2019 |
Shimell JJ, Shah BS, Cain SM, Thouta S, Kuhlmann N, Tatarnikov I, Jovellar DB, Brigidi GS, Kass J, Milnerwood AJ, Snutch TP, Bamji SX. The X-Linked Intellectual Disability Gene Zdhhc9 Is Essential for Dendrite Outgrowth and Inhibitory Synapse Formation. Cell Reports. 29: 2422-2437.e8. PMID 31747610 DOI: 10.1016/J.Celrep.2019.10.065 |
0.318 |
|
| 2019 |
Marks WN, Zabder NK, Greba Q, Cain SM, Snutch TP, Howland JG. The T-type calcium channel blocker Z944 reduces conditioned fear in Genetic Absence Epilepsy Rats from Strasbourg and the Non-Epileptic Control strain. The European Journal of Neuroscience. PMID 30889299 DOI: 10.1111/Ejn.14406 |
0.503 |
|
| 2019 |
Loonen ICM, Jansen NA, Cain SM, Schenke M, Voskuyl RA, Yung AC, Bohnet B, Kozlowski P, Thijs RD, Ferrari MD, Snutch TP, van den Maagdenberg AMJM, Tolner EA. Brainstem spreading depolarization and cortical dynamics during fatal seizures in Cacna1a S218L mice. Brain : a Journal of Neurology. PMID 30649209 DOI: 10.1093/Brain/Awy325 |
0.403 |
|
| 2018 |
Marks WN, Zabder NK, Cain SM, Snutch TP, Howland JG. The T-type calcium channel antagonist, Z944, alters social behavior in Genetic Absence Epilepsy Rats from Strasbourg. Behavioural Brain Research. 361: 54-64. PMID 30550952 DOI: 10.1016/J.Bbr.2018.12.021 |
0.468 |
|
| 2018 |
Garcia-Caballero A, Zhang FX, Hodgkinson V, Huang J, Chen L, Souza IA, Cain S, Kass J, Alles S, Snutch TP, Zamponi GW. T-type calcium channels functionally interact with spectrin (α/β) and ankyrin B. Molecular Brain. 11: 24. PMID 29720258 DOI: 10.1186/S13041-018-0368-5 |
0.484 |
|
| 2018 |
Cain SM, Tyson JR, Choi HB, Ko R, Lin PJC, LeDue JM, Powell KL, Bernier LP, Rungta RL, Yang Y, Cullis PR, O'Brien TJ, MacVicar BA, Snutch TP. Ca3.2 drives sustained burst-firing, which is critical for absence seizure propagation in reticular thalamic neurons. Epilepsia. PMID 29468672 DOI: 10.1111/Epi.14018 |
0.546 |
|
| 2017 |
Proft J, Rzhepetskyy Y, Lazniewska J, Zhang FX, Cain SM, Snutch TP, Zamponi GW, Weiss N. The Cacna1h mutation in the GAERS model of absence epilepsy enhances T-type Ca(2+) currents by altering calnexin-dependent trafficking of Cav3.2 channels. Scientific Reports. 7: 11513. PMID 28912545 DOI: 10.1038/S41598-017-11591-5 |
0.605 |
|
| 2017 |
Cain SM, Garcia E, Waheed Z, Jones KL, Bushell TJ, Snutch TP. GABAB Receptors Suppress Burst-Firing in Reticular Thalamic Neurons. Channels (Austin, Tex.). 0. PMID 28742985 DOI: 10.1080/19336950.2017.1358836 |
0.523 |
|
| 2017 |
Henbid MT, Marks WN, Collins MJ, Cain SM, Snutch TP, Howland JG. Sociability impairments in Genetic Absence Epilepsy Rats from Strasbourg: Reversal by the T-type calcium channel antagonist Z944. Experimental Neurology. PMID 28658605 DOI: 10.1016/J.Expneurol.2017.06.022 |
0.543 |
|
| 2017 |
Cain SM, Bohnet B, LeDue J, Yung AC, Garcia E, Tyson JR, Alles SR, Han H, van den Maagdenberg AM, Kozlowski P, MacVicar BA, Snutch TP. In vivo imaging reveals that pregabalin inhibits cortical spreading depression and propagation to subcortical brain structures. Proceedings of the National Academy of Sciences of the United States of America. PMID 28223480 DOI: 10.1073/Pnas.1614447114 |
0.401 |
|
| 2016 |
Cain SM, Ahn S, Garcia E, Zhang Y, Waheed Z, Tyson JR, Yang Y, Van Sung T, Phillips AG, Snutch TP. Heantos-4, a natural plant extract used in the treatment of drug addiction, modulates T-type calcium channels and thalamocortical burst-firing. Molecular Brain. 9: 94. PMID 27919294 DOI: 10.1186/S13041-016-0274-7 |
0.577 |
|
| 2016 |
Marks WN, Greba Q, Cain SM, Snutch TP, Howland JG. The T-type calcium channel antagonist Z944 disrupts prepulse inhibition in both epileptic and non-epileptic rats. Neuroscience. PMID 27365170 DOI: 10.1016/J.Neuroscience.2016.06.035 |
0.546 |
|
| 2016 |
Marks WN, Cain SM, Snutch TP, Howland JG. The T-type calcium channel antagonist Z944 rescues impairments in crossmodal and visual recognition memory in Genetic Absence Epilepsy Rats from Strasbourg. Neurobiology of Disease. PMID 27282256 DOI: 10.1016/J.Nbd.2016.06.001 |
0.462 |
|
| 2015 |
Marks WN, Cavanagh ME, Greba Q, Cain SM, Snutch TP, Howland JG. The GAERS model of absence epilepsy exhibits alterations in fear conditioning and latent inhibition consistent with psychiatric comorbidities in humans. The European Journal of Neuroscience. PMID 26490879 DOI: 10.1111/Ejn.13110 |
0.346 |
|
| 2015 |
Rungta RL, Choi HB, Tyson JR, Malik A, Dissing-Olesen L, Lin PJ, Cain SM, Cullis PR, Snutch TP, MacVicar BA. The cellular mechanisms of neuronal swelling underlying cytotoxic edema. Cell. 161: 610-21. PMID 25910210 DOI: 10.1016/J.Cell.2015.03.029 |
0.343 |
|
| 2015 |
Cain SM, Tyson JR, Jones KL, Snutch TP. Thalamocortical neurons display suppressed burst-firing due to an enhanced Ih current in a genetic model of absence epilepsy. PflüGers Archiv : European Journal of Physiology. 467: 1367-82. PMID 24953239 DOI: 10.1007/S00424-014-1549-4 |
0.498 |
|
| 2014 |
Ranzato E, Magnelli V, Martinotti S, Waheed Z, Cain SM, Snutch TP, Marchetti C, Burlando B. Epigallocatechin-3-gallate elicits Ca2+ spike in MCF-7 breast cancer cells: essential role of Cav3.2 channels. Cell Calcium. 56: 285-95. PMID 25260713 DOI: 10.1016/J.Ceca.2014.09.002 |
0.432 |
|
| 2014 |
Powell KL, Cain SM, Snutch TP, O'Brien TJ. Low threshold T-type calcium channels as targets for novel epilepsy treatments. British Journal of Clinical Pharmacology. 77: 729-39. PMID 23834404 DOI: 10.1111/Bcp.12205 |
0.574 |
|
| 2014 |
Cain SM, Hildebr ME, Snutch TP. T-type calcium channels and epilepsy Pathologies of Calcium Channels. 77-96. DOI: 10.1007/978-3-642-40282-1_4 |
0.525 |
|
| 2013 |
Cain SM, Snutch TP. T-type calcium channels in burst-firing, network synchrony, and epilepsy. Biochimica Et Biophysica Acta. 1828: 1572-8. PMID 22885138 DOI: 10.1016/J.Bbamem.2012.07.028 |
0.568 |
|
| 2012 |
Tringham E, Powell KL, Cain SM, Kuplast K, Mezeyova J, Weerapura M, Eduljee C, Jiang X, Smith P, Morrison JL, Jones NC, Braine E, Rind G, Fee-Maki M, Parker D, et al. T-type calcium channel blockers that attenuate thalamic burst firing and suppress absence seizures. Science Translational Medicine. 4: 121ra19. PMID 22344687 DOI: 10.1126/Scitranslmed.3003120 |
0.578 |
|
| 2011 |
Cain SM, Snutch TP. Voltage-gated calcium channels and disease. Biofactors (Oxford, England). 37: 197-205. PMID 21698699 DOI: 10.1002/Biof.158 |
0.481 |
|
| 2010 |
Cain SM, Snutch TP. Contributions of T-type calcium channel isoforms to neuronal firing. Channels (Austin, Tex.). 4: 475-82. PMID 21139420 DOI: 10.4161/Chan.4.6.14106 |
0.566 |
|
| 2010 |
David LS, Garcia E, Cain SM, Thau E, Tyson JR, Snutch TP. Splice-variant changes of the Ca(V)3.2 T-type calcium channel mediate voltage-dependent facilitation and associate with cardiac hypertrophy and development. Channels (Austin, Tex.). 4: 375-89. PMID 20699644 DOI: 10.4161/Chan.4.5.12874 |
0.433 |
|
| 2010 |
Cain SM, Snutch TP. Voltage-gated calcium channels in epilepsy Epilepsia. 51: 11. DOI: 10.1111/J.1528-1167.2010.02797.X |
0.551 |
|
| 2009 |
Powell KL, Cain SM, Ng C, Sirdesai S, David LS, Kyi M, Garcia E, Tyson JR, Reid CA, Bahlo M, Foote SJ, Snutch TP, O'Brien TJ. A Cav3.2 T-type calcium channel point mutation has splice-variant-specific effects on function and segregates with seizure expression in a polygenic rat model of absence epilepsy. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 29: 371-80. PMID 19144837 DOI: 10.1523/Jneurosci.5295-08.2009 |
0.541 |
|
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