Joseph Chavarria-Smith - Publications
Affiliations: | 2010-2015 | University of California, Berkeley, Berkeley, CA, United States |
Year | Citation | Score | |||
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2016 | Chavarría-Smith J, Mitchell PS, Ho AM, Daugherty MD, Vance RE. Functional and Evolutionary Analyses Identify Proteolysis as a General Mechanism for NLRP1 Inflammasome Activation. Plos Pathogens. 12: e1006052. PMID 27926929 DOI: 10.1371/Journal.Ppat.1006052 | 0.681 | |||
2015 | Chavarría-Smith J, Vance RE. The NLRP1 inflammasomes. Immunological Reviews. 265: 22-34. PMID 25879281 DOI: 10.1111/Imr.12283 | 0.716 | |||
2014 | Ewald SE, Chavarria-Smith J, Boothroyd JC. NLRP1 is an inflammasome sensor for Toxoplasma gondii. Infection and Immunity. 82: 460-8. PMID 24218483 DOI: 10.1128/Iai.01170-13 | 0.535 | |||
2013 | Rayamajhi M, Zak DE, Chavarria-Smith J, Vance RE, Miao EA. Cutting edge: Mouse NAIP1 detects the type III secretion system needle protein. Journal of Immunology (Baltimore, Md. : 1950). 191: 3986-9. PMID 24043898 DOI: 10.4049/Jimmunol.1301549 | 0.613 | |||
2013 | Chavarría-Smith J, Vance RE. Direct proteolytic cleavage of NLRP1B is necessary and sufficient for inflammasome activation by anthrax lethal factor. Plos Pathogens. 9: e1003452. PMID 23818853 DOI: 10.1371/Journal.Ppat.1003452 | 0.703 | |||
2013 | von Moltke J, Ayres JS, Kofoed EM, Chavarría-Smith J, Vance RE. Recognition of bacteria by inflammasomes. Annual Review of Immunology. 31: 73-106. PMID 23215645 DOI: 10.1146/Annurev-Immunol-032712-095944 | 0.42 | |||
2011 | Kuo TC, Chavarria-Smith JE, Huang D, Schlissel MS. Forced expression of cyclin-dependent kinase 6 confers resistance of pro-B acute lymphocytic leukemia to Gleevec treatment. Molecular and Cellular Biology. 31: 2566-76. PMID 21536647 DOI: 10.1128/Mcb.01349-10 | 0.531 | |||
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