James J. Collins, PhD
Affiliations: | Biological Engineering | Massachusetts Institute of Technology, Cambridge, MA, United States |
Area:
Systems Biology, Synthetic BiologyWebsite:
http://collinslab.mit.edu/Google:
"James Collins"Bio:
https://en.wikipedia.org/wiki/James_Collins_(bioengineer)
Cross-listing: Chemistry Tree - Neurotree - Cell Biology Tree
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Publications
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Ofori-Anyinam B, Hamblin M, Coldren ML, et al. (2024) Catalase activity deficiency sensitizes multidrug-resistant Mycobacterium tuberculosis to the ATP synthase inhibitor bedaquiline. Nature Communications. 15: 9792 |
Kuru E, Rittichier J, de Puig H, et al. (2024) Rapid discovery and evolution of nanosensors containing fluorogenic amino acids. Nature Communications. 15: 7531 |
Wong F, de la Fuente-Nunez C, Collins JJ. (2023) Leveraging artificial intelligence in the fight against infectious diseases. Science (New York, N.Y.). 381: 164-170 |
Valeri JA, Soenksen LR, Collins KM, et al. (2023) BioAutoMATED: An end-to-end automated machine learning tool for explanation and design of biological sequences. Cell Systems. 14: 525-542.e9 |
Phillips EA, Silverman AD, Joneja A, et al. (2023) Detection of viral RNAs at ambient temperature via reporter proteins produced through the target-splinted ligation of DNA probes. Nature Biomedical Engineering |
Cabera A, Edelstein HI, Glykofrydis F, et al. (2022) The sound of silence: Transgene silencing in mammalian cell engineering. Cell Systems. 13: 950-973 |
Sun Q, Vega NM, Cervantes B, et al. (2022) Enhancing nutritional niche and host defenses by modifying the gut microbiome. Molecular Systems Biology. 18: e9933 |
Warrier T, El Farran C, Zeng Y, et al. (2022) SETDB1 acts as a topological accessory to Cohesin via an H3K9me3-independent, genomic shunt for regulating cell fates. Nucleic Acids Research |
Lummertz da Rocha E, Kubaczka C, Sugden WW, et al. (2022) CellComm infers cellular crosstalk that drives haematopoietic stem and progenitor cell development. Nature Cell Biology |
Lobritz MA, Andrews IW, Braff D, et al. (2021) Increased energy demand from anabolic-catabolic processes drives β-lactam antibiotic lethality. Cell Chemical Biology |