BETA: Related publications
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| Moonen JR, Chappell J, Shi M, et al. (2022) KLF4 recruits SWI/SNF to increase chromatin accessibility and reprogram the endothelial enhancer landscape under laminar shear stress. Nature Communications. 13: 4941 |
| Kim DS, Risca VI, Reynolds DL, et al. (2021) The dynamic, combinatorial cis-regulatory lexicon of epidermal differentiation. Nature Genetics |
| Hennigs JK, Cao A, Li CG, et al. (2020) PPARγ-p53-Mediated Vasculoregenerative Program to Reverse Pulmonary Hypertension. Circulation Research |
| van der Feen DE, Bossers GPL, Hagdorn QAJ, et al. (2020) Cellular senescence impairs the reversibility of pulmonary arterial hypertension. Science Translational Medicine. 12 |
| Tian W, Jiang X, Sung YK, et al. (2019) Phenotypically-Silent Bone Morphogenetic Protein Receptor 2 (Bmpr2) Mutations Predispose Rats to Inflammation-Induced Pulmonary Arterial Hypertension by Enhancing The Risk for Neointimal Transformation. Circulation |
| Sailani MR, Chappell J, Inlora J, et al. (2017) WISP3 mutation associated with Pseudorheumatoid Dysplasia. Cold Spring Harbor Molecular Case Studies |
| Gu M, Shao NY, Sa S, et al. (2016) Patient-Specific iPSC-Derived Endothelial Cells Uncover Pathways that Protect against Pulmonary Hypertension in BMPR2 Mutation Carriers. Cell Stem Cell |
| Sa S, Gu M, Chappell J, et al. (2016) iPSC Model of Pulmonary Arterial Hypertension Reveals Novel Gene Expression and Patient Specificity. American Journal of Respiratory and Critical Care Medicine |
| Singh AM, Chappell J, Trost R, et al. (2013) Cell-cycle control of developmentally regulated transcription factors accounts for heterogeneity in human pluripotent cells. Stem Cell Reports. 1: 532-44 |
| Chappell J, Dalton S. (2013) Roles for MYC in the establishment and maintenance of pluripotency. Cold Spring Harbor Perspectives in Medicine. 3: a014381 |