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Cordelia J. Barrick, Ph.D. - Publications

Affiliations: 
2007 Toxicology University of North Carolina, Chapel Hill, Chapel Hill, NC 
Area:
Toxicology
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10 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2015 Crowley JJ, Zhabotynsky V, Sun W, Huang S, Pakatci IK, Kim Y, Wang JR, Morgan AP, Calaway JD, Aylor DL, Yun Z, Bell TA, Buus RJ, Calaway ME, Didion JP, ... ... Barrick CJ, et al. Corrigendum: Analyses of allele-specific gene expression in highly divergent mouse crosses identifies pervasive allelic imbalance. Nature Genetics. 47: 690. PMID 26018903 DOI: 10.1038/Ng0615-690A  0.579
2015 Crowley JJ, Zhabotynsky V, Sun W, Huang S, Pakatci IK, Kim Y, Wang JR, Morgan AP, Calaway JD, Aylor DL, Yun Z, Bell TA, Buus RJ, Calaway ME, Didion JP, ... ... Barrick CJ, et al. Analyses of allele-specific gene expression in highly divergent mouse crosses identifies pervasive allelic imbalance. Nature Genetics. 47: 353-60. PMID 25730764 DOI: 10.1038/Ng.3222  0.613
2014 Crowley JJ, Kim Y, Lenarcic AB, Quackenbush CR, Barrick CJ, Adkins DE, Shaw GS, Miller DR, de Villena FP, Sullivan PF, Valdar W. Genetics of adverse reactions to haloperidol in a mouse diallel: a drug-placebo experiment and Bayesian causal analysis. Genetics. 196: 321-47. PMID 24240528 DOI: 10.1534/Genetics.113.156901  0.326
2013 Lenhart PM, Broselid S, Barrick CJ, Leeb-Lundberg LM, Caron KM. G-protein-coupled receptor 30 interacts with receptor activity-modifying protein 3 and confers sex-dependent cardioprotection. Journal of Molecular Endocrinology. 51: 191-202. PMID 23674134 DOI: 10.1530/Jme-13-0021  0.337
2012 Yang F, Dong A, Mueller P, Caicedo J, Sutton AM, Odetunde J, Barrick CJ, Klyachkin YM, Abdel-Latif A, Smyth SS. Coronary artery remodeling in a model of left ventricular pressure overload is influenced by platelets and inflammatory cells. Plos One. 7: e40196. PMID 22916095 DOI: 10.1371/Journal.Pone.0040196  0.349
2012 Barrick CJ, Lenhart PM, Dackor RT, Nagle E, Caron KM. Loss of receptor activity-modifying protein 3 exacerbates cardiac hypertrophy and transition to heart failure in a sex-dependent manner. Journal of Molecular and Cellular Cardiology. 52: 165-74. PMID 22100352 DOI: 10.1016/J.Yjmcc.2011.10.021  0.505
2009 Barrick CJ, Dong A, Waikel R, Corn D, Yang F, Threadgill DW, Smyth SS. Parent-of-origin effects on cardiac response to pressure overload in mice. American Journal of Physiology. Heart and Circulatory Physiology. 297: H1003-9. PMID 19561308 DOI: 10.1152/Ajpheart.00896.2008  0.612
2009 Barrick CJ, Roberts RB, Rojas M, Rajamannan NM, Suitt CB, O'Brien KD, Smyth SS, Threadgill DW. Reduced EGFR causes abnormal valvular differentiation leading to calcific aortic stenosis and left ventricular hypertrophy in C57BL/6J but not 129S1/SvImJ mice. American Journal of Physiology. Heart and Circulatory Physiology. 297: H65-75. PMID 19448146 DOI: 10.1152/Ajpheart.00866.2008  0.618
2008 Barrick CJ, Yu M, Chao HH, Threadgill DW. Chronic pharmacologic inhibition of EGFR leads to cardiac dysfunction in C57BL/6J mice. Toxicology and Applied Pharmacology. 228: 315-25. PMID 18313710 DOI: 10.1016/J.Taap.2007.12.012  0.634
2007 Barrick CJ, Rojas M, Schoonhoven R, Smyth SS, Threadgill DW. Cardiac response to pressure overload in 129S1/SvImJ and C57BL/6J mice: temporal- and background-dependent development of concentric left ventricular hypertrophy. American Journal of Physiology. Heart and Circulatory Physiology. 292: H2119-30. PMID 17172276 DOI: 10.1152/Ajpheart.00816.2006  0.619
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