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Faye Boeckman - Publications

Affiliations: 
Life Technologies, Carlsbad, CA, United States 
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7 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
1999 Rohl CA, Boeckman FA, Baker C, Scheuer T, Catterall WA, Klevit RE. Solution structure of the sodium channel inactivation gate. Biochemistry. 38: 855-61. PMID 9893979 DOI: 10.1021/Bi9823380  0.439
1997 Brimecombe JC, Boeckman FA, Aizenman E. Functional consequences of NR2 subunit composition in single recombinant N-methyl-D-aspartate receptors. Proceedings of the National Academy of Sciences of the United States of America. 94: 11019-24. PMID 9380752 DOI: 10.1073/Pnas.94.20.11019  0.543
1997 Blanpied TA, Boeckman FA, Aizenman E, Johnson JW. Trapping channel block of NMDA-activated responses by amantadine and memantine. Journal of Neurophysiology. 77: 309-23. PMID 9120573 DOI: 10.1152/Jn.1997.77.1.309  0.776
1997 Sinor JD, Boeckman FA, Aizenman E. Intrinsic redox properties of N-methyl-D-aspartate receptor can determine the developmental expression of excitotoxicity in rat cortical neurons in vitro. Brain Research. 747: 297-303. PMID 9046005 DOI: 10.1016/S0006-8993(96)01237-1  0.668
1996 Boeckman FA, Aizenman E. Pharmacological properties of acquired excitotoxicity in Chinese hamster ovary cells transfected with N-methyl-D-aspartate receptor subunits. The Journal of Pharmacology and Experimental Therapeutics. 279: 515-23. PMID 8930153  0.782
1994 Boeckman FA, Aizenman E. Stable transfection of the NR1 subunit in Chinese hamster ovary cells fails to produce a functional N-methyl-D-aspartate receptor. Neuroscience Letters. 173: 189-92. PMID 7936412 DOI: 10.1016/0304-3940(94)90180-5  0.758
1992 Aizenman E, Boeckman FA, Rosenberg PA. Glutathione prevents 2,4,5-trihydroxyphenylalanine excitotoxicity by maintaining it in a reduced, non-active form. Neuroscience Letters. 144: 233-6. PMID 1436708 DOI: 10.1016/0304-3940(92)90757-X  0.666
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