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Rachel Tanos, Ph.D. - Publications

Affiliations: 
2012 Biochemistry and Molecular Biology Pennsylvania State University, State College, PA, United States 
Area:
Biochemistry, Molecular Biology
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5 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2019 Muku GE, Kusnadi A, Kuzu G, Tanos R, Murray IA, Gowda K, Amin S, Perdew GH. Selective Ah receptor modulators attenuate NPC1L1-mediated cholesterol uptake through repression of SREBP-2 transcriptional activity. Laboratory Investigation; a Journal of Technical Methods and Pathology. PMID 31417158 DOI: 10.1038/S41374-019-0306-X  0.652
2012 Tanos R, Murray IA, Smith PB, Patterson A, Perdew GH. Role of the Ah receptor in homeostatic control of fatty acid synthesis in the liver. Toxicological Sciences : An Official Journal of the Society of Toxicology. 129: 372-9. PMID 22696238 DOI: 10.1093/Toxsci/Kfs204  0.621
2012 Tanos R, Patel RD, Murray IA, Smith PB, Patterson AD, Perdew GH. Aryl hydrocarbon receptor regulates the cholesterol biosynthetic pathway in a dioxin response element-independent manner. Hepatology (Baltimore, Md.). 55: 1994-2004. PMID 22234961 DOI: 10.1002/Hep.25571  0.665
2011 Smith KJ, Murray IA, Tanos R, Tellew J, Boitano AE, Bisson WH, Kolluri SK, Cooke MP, Perdew GH. Identification of a high-affinity ligand that exhibits complete aryl hydrocarbon receptor antagonism. The Journal of Pharmacology and Experimental Therapeutics. 338: 318-27. PMID 21493753 DOI: 10.1124/Jpet.110.178392  0.644
2011 Murray IA, Flaveny CA, Chiaro CR, Sharma AK, Tanos RS, Schroeder JC, Amin SG, Bisson WH, Kolluri SK, Perdew GH. Suppression of cytokine-mediated complement factor gene expression through selective activation of the Ah receptor with 3',4'-dimethoxy-α-naphthoflavone. Molecular Pharmacology. 79: 508-19. PMID 21127131 DOI: 10.1124/Mol.110.069369  0.523
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