Donald W. Hilgemann
Affiliations: | Physiology | University of Texas Southwestern Medical Center, Dallas, TX, United States |
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"Donald Hilgemann"Parents
Sign in to add mentor| Glenn A. Langer | grad student | UCLA - School of Medicine (Computational Biology Tree) |
Children
Sign in to add trainee| Kyle B. Womack | grad student | 1997-2000 | University of Texas Southwester Medical Center |
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Publications
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| Deisl C, Hilgemann DW, Syeda R, et al. (2021) TMEM16F and dynamins control expansive plasma membrane reservoirs. Nature Communications. 12: 4990 |
| Hilgemann DW. (2020) Regulation of ion transport from within ion transit pathways. The Journal of General Physiology. 152 |
| Hilgemann DW. (2020) Control of cardiac contraction by sodium: Promises, reckonings, and new beginnings. Cell Calcium. 85: 102129 |
| Deisl C, Fine M, Moe OW, et al. (2019) Hypertrophy of human embryonic stem cell-derived cardiomyocytes supported by positive feedback between Ca and diacylglycerol signals. Pflugers Archiv : European Journal of Physiology |
| Hilgemann DW, Lin MJ, Fine M, et al. (2019) On the existence of endocytosis driven by membrane phase separations. Biochimica Et Biophysica Acta. Biomembranes |
| Bricogne C, Fine M, Pereira PM, et al. (2019) Author Correction: TMEM16F activation by Ca triggers plasma membrane expansion and directs PD-1 trafficking. Scientific Reports. 9: 7705 |
| Bricogne C, Fine M, Pereira PM, et al. (2019) TMEM16F activation by Ca triggers plasma membrane expansion and directs PD-1 trafficking. Scientific Reports. 9: 619 |
| Hilgemann DW, Dai G, Collins A, et al. (2018) Correction: Lipid signaling to membrane proteins: From second messengers to membrane domains and adapter-free endocytosis. The Journal of General Physiology |
| Hilgemann DW, Dai G, Collins A, et al. (2018) Lipid signaling to membrane proteins: From second messengers to membrane domains and adapter-free endocytosis. The Journal of General Physiology |
| Lu FM, Hilgemann DW. (2017) Na/K pump inactivation, subsarcolemmal Na measurements, and cytoplasmic ion turnover kinetics contradict restricted Na spaces in murine cardiac myocytes. The Journal of General Physiology |