John D. Mably
Affiliations: | Harvard University, Cambridge, MA, United States |
Area:
zebrafishGoogle:
"John Mably"Cross-listing: DevTree
BETA: Related publications
See more...
Publications
|
You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect. |
| Wang Y, Zhang M, Wang R, et al. (2024) Therapeutic Inhibition of Protects Against Cardiac Hypertrophy. Circulation Research |
| He X, Yang T, Lu YW, et al. (2024) The long non-coding RNA CARDINAL attenuates cardiac hypertrophy by modulating protein translation. The Journal of Clinical Investigation |
| Mably JD, Wang DZ. (2023) Long non-coding RNAs in cardiac hypertrophy and heart failure: functions, mechanisms and clinical prospects. Nature Reviews. Cardiology |
| Lu YW, Liang Z, Guo H, et al. (2023) PCBP1 regulates alternative splicing of AARS2 in congenital cardiomyopathy. Biorxiv : the Preprint Server For Biology |
| Gao F, Liang T, Lu YW, et al. (2023) A defect in mitochondrial protein translation influences mitonuclear communication in the heart. Nature Communications. 14: 1595 |
| Kawasaki J, Aegerter S, Fevurly RD, et al. (2014) RASA1 functions in EPHB4 signaling pathway to suppress endothelial mTORC1 activity. The Journal of Clinical Investigation. 124: 2774-84 |
| Akbareian SE, Nagy N, Steiger CE, et al. (2013) Enteric neural crest-derived cells promote their migration by modifying their microenvironment through tenascin-C production. Developmental Biology. 382: 446-56 |
| Sah R, Mesirca P, Van den Boogert M, et al. (2013) Ion channel-kinase TRPM7 is required for maintaining cardiac automaticity. Proceedings of the National Academy of Sciences of the United States of America. 110: E3037-46 |
| Rosen JN, Sogah VM, Ye LY, et al. (2013) ccm2-like is required for cardiovascular development as a novel component of the Heg-CCM pathway. Developmental Biology. 376: 74-85 |
| Sah R, Mesirca P, Mason X, et al. (2013) The Ion Channel-Kinase, TRPM7, is Required for Cardiac Automaticity Biophysical Journal. 104: 379a |