Julius C. Barsi, Ph.D.

2019- Systems Developmental Biology The Bermuda Institute of Ocean Sciences 
molecular biology, cis-regulatory code, comprehensive GRNs, bauplan
"Julius Christopher Barsi"

Julius was born in Pasadena, a suburb of Los Angeles California, in 1977. His wanderlust led him to complete undergraduate studies abroad. He returned to the States after having spent several years in Patagonia. As a young man, he moved to Austin to pursue doctoral research at the University of Texas. In 2005 he published his first research paper on mammalian development and received his Doctor of Philosophy diploma in molecular biology the following year. He returned to his hometown of Pasadena to complete postdoctoral training at the California Institute of Technology, where he was promoted to the rank of Senior Fellow. In 2019 the Barsi lab opened its doors at the Bermuda Institute of Ocean Sciences.


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Karen Artzt grad student 2006 UT Austin
 (A core signaling component of the Notch network + a molecular interaction database accessible through an online VLSIC-like interface.)
Eric H. Davidson post-doc 2008-2015 Caltech
 (Cis-regulatory control of the initial neurogenic pattern of onecut gene expression in the sea urchin embryo)
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Choi HM, Calvert CR, Husain N, et al. (2016) Mapping a multiplexed zoo of mRNA expression. Development (Cambridge, England). 143: 3632-3637
Barsi JC, Davidson EH. (2015) cis-Regulatory control of the initial neurogenic pattern of onecut gene expression in the sea urchin embryo. Developmental Biology
Barsi JC, Tu Q, Calestani C, et al. (2015) Genome-wide assessment of differential effector gene use in embryogenesis. Development (Cambridge, England)
Barsi JC, Li E, Davidson EH. (2015) Geometric control of ciliated band regulatory states in the sea urchin embryo. Development (Cambridge, England). 142: 953-61
Barsi JC, Tu Q, Davidson EH. (2014) General approach for in vivo recovery of cell type-specific effector gene sets. Genome Research. 24: 860-8
Wu JI, Rajendra R, Barsi JC, et al. (2007) Targeted disruption of Mib2 causes exencephaly with a variable penetrance. Genesis (New York, N.Y. : 2000). 45: 722-7
Barsi JC, Rajendra R, Wu JI, et al. (2005) Mind bomb1 is a ubiquitin ligase essential for mouse embryonic development and Notch signaling. Mechanisms of Development. 122: 1106-17
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