Andrew F James
Affiliations: | 2000-2001 | Cardiovascular Research | Kings College London, London, England, United Kingdom |
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"Andrew James"
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| Cheng H, Charles I, James AF, et al. (2022) Delayed Ventricular Repolarization and Sodium Channel Current Modification in a Mouse Model of Rett Syndrome. International Journal of Molecular Sciences. 23 |
| Caves RE, Carpenter A, Choisy SC, et al. (2020) Inhibition of voltage-gated Na currents by eleclazine in rat atrial and ventricular myocytes. Heart Rhythm O2. 1: 206-214 |
| Bond RC, Choisy SC, Bryant SM, et al. (2020) Ion currents, action potentials, and noradrenergic responses in rat pulmonary vein and left atrial cardiomyocytes. Physiological Reports. 8: e14432 |
| Caves RE, Cheng H, Choisy SC, et al. (2017) Atrial-ventricular differences in rabbit cardiac voltage-gated Na(+) currents: basis for atrial-selective block by ranolazine. Heart Rhythm |
| Cheng H, Li J, James AF, et al. (2016) Characterization and influence of cardiac background sodium current in the atrioventricular node. Journal of Molecular and Cellular Cardiology |
| Choisy SC, Cheng H, Orchard CH, et al. (2015) Electrophysiological properties of myocytes isolated from the mouse atrioventricular node: L-type ICa, IKr, If, and Na-Ca exchange. Physiological Reports. 3 |
| Gadeberg HC, Bryant SM, James AF, et al. (2015) Altered Na/Ca exchange distribution and activity in ventricular myocytes from failing hearts. American Journal of Physiology. Heart and Circulatory Physiology. ajpheart.00597.2015 |
| Hancock JM, Weatherall KL, Choisy SC, et al. (2015) Selective activation of heteromeric SK channels contributes to action potential repolarization in mouse atrial myocytes. Heart Rhythm : the Official Journal of the Heart Rhythm Society. 12: 1003-15 |
| Bond RC, Choisy SC, Bryant SM, et al. (2014) Inhibition of a TREK-like K+ channel current by noradrenaline requires both β1- and β2-adrenoceptors in rat atrial myocytes. Cardiovascular Research. 104: 206-15 |
| Kim SJ, Zhang H, Khaliulin I, et al. (2012) Activation of glibenclamide-sensitive ATP-sensitive K+ channels during β-adrenergically induced metabolic stress produces a substrate for atrial tachyarrhythmia Circulation: Arrhythmia and Electrophysiology. 5: 1184-1192 |