| Year |
Citation |
Score |
| 2023 |
Renz C, Asimaki E, Meister C, Albanèse V, Petriukov K, Krapoth NC, Wegmann S, Wollscheid HP, Wong RP, Fulzele A, Chen JX, Léon S, Ulrich HD. Ubiquiton-An inducible, linkage-specific polyubiquitylation tool. Molecular Cell. PMID 38103558 DOI: 10.1016/j.molcel.2023.11.016 |
0.65 |
|
| 2023 |
Yakoub G, Choi YS, Wong RP, Strauch T, Ann KJ, Cohen RE, Ulrich HD. Avidity-based biosensors for ubiquitylated PCNA reveal choreography of DNA damage bypass. Science Advances. 9: eadf3041. PMID 37672592 DOI: 10.1126/sciadv.adf3041 |
0.792 |
|
| 2021 |
Cabello-Lobato MJ, González-Garrido C, Cano-Linares MI, Wong RP, Yáñez-Vílchez A, Morillo-Huesca M, Roldán-Romero JM, Vicioso M, González-Prieto R, Ulrich HD, Prado F. Physical interactions between MCM and Rad51 facilitate replication fork lesion bypass and ssDNA gap filling by non-recombinogenic functions. Cell Reports. 36: 109440. PMID 34320356 DOI: 10.1016/j.celrep.2021.109440 |
0.757 |
|
| 2021 |
Wong RP, Petriukov K, Ulrich HD. Daughter-strand gaps in DNA replication - substrates of lesion processing and initiators of distress signalling. Dna Repair. 105: 103163. PMID 34186497 DOI: 10.1016/j.dnarep.2021.103163 |
0.812 |
|
| 2019 |
Wong RP, García-Rodríguez N, Zilio N, Hanulová M, Ulrich HD. Processing of DNA Polymerase-Blocking Lesions during Genome Replication Is Spatially and Temporally Segregated from Replication Forks. Molecular Cell. PMID 31607544 DOI: 10.1016/J.Molcel.2019.09.015 |
0.748 |
|
| 2018 |
García-Rodríguez N, Wong RP, Ulrich HD. The helicase Pif1 functions in the template switching pathway of DNA damage bypass. Nucleic Acids Research. PMID 30107417 DOI: 10.1093/Nar/Gky648 |
0.82 |
|
| 2018 |
García-Rodríguez N, Morawska M, Wong RP, Daigaku Y, Ulrich HD. Spatial separation between replisome- and template-induced replication stress signaling. The Embo Journal. PMID 29581097 DOI: 10.15252/Embj.201798369 |
0.682 |
|
| 2017 |
Hung SH, Wong RP, Ulrich HD, Kao CF. Monoubiquitylation of histone H2B contributes to the bypass of DNA damage during and after DNA replication. Proceedings of the National Academy of Sciences of the United States of America. PMID 28246327 DOI: 10.1073/Pnas.1612633114 |
0.816 |
|
| 2016 |
García-Rodríguez N, Wong RP, Ulrich HD. Functions of Ubiquitin and SUMO in DNA Replication and Replication Stress. Frontiers in Genetics. 7: 87. PMID 27242895 DOI: 10.3389/Fgene.2016.00087 |
0.826 |
|
| 2011 |
Wong RP, Lin H, Khosravi S, Piche B, Jafarnejad SM, Chen DW, Li G. Tumour suppressor ING1b maintains genomic stability upon replication stress. Nucleic Acids Research. 39: 3632-42. PMID 21227930 DOI: 10.1093/nar/gkq1337 |
0.509 |
|
| 2010 |
Wong RP, Lin H, Chen DW, Li G. Abstract 2979: Tumor suppressor ING1b maintains genomic stability through PCNA monoubiquitination and histone H4 acetylation Cancer Research. 70: 2979-2979. DOI: 10.1158/1538-7445.Am10-2979 |
0.568 |
|
| 2007 |
Kuo WH, Wang Y, Wong RP, Campos EI, Li G. The ING1b tumor suppressor facilitates nucleotide excision repair by promoting chromatin accessibility to XPA. Experimental Cell Research. 313: 1628-38. PMID 17379210 DOI: 10.1016/j.yexcr.2007.02.010 |
0.575 |
|
| Show low-probability matches. |