Marina P. di Magliano

Affiliations: 
Cellular and Molecular Biology University of Michigan, Ann Arbor, Ann Arbor, MI 
Area:
Cell Biology, Oncology
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"Marina di Magliano"
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Publications

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Lu S, Kim HS, Cao Y, et al. (2023) KMT2D links TGF-β signaling to noncanonical activin pathway and regulates pancreatic cancer cell plasticity. International Journal of Cancer
Carpenter ES, Elhossiny AM, Kadiyala P, et al. (2023) Analysis of donor pancreata defines the transcriptomic signature and microenvironment of early pre-neoplastic pancreatic lesions. Biorxiv : the Preprint Server For Biology
Yan W, Steele NG, Kemp SB, et al. (2023) Notch signaling regulates immunosuppressive tumor-associated macrophage function in pancreatic cancer. Biorxiv : the Preprint Server For Biology
Mathison AJ, Kerketta R, de Assuncao TM, et al. (2021) Kras induces changes in chromatin territories that differentially impact early nuclear reprogramming in pancreatic cells. Genome Biology. 22: 289
Kremer DM, Nelson BS, Lin L, et al. (2021) GOT1 inhibition promotes pancreatic cancer cell death by ferroptosis. Nature Communications. 12: 4860
Azizi N, Toma J, Martin M, et al. (2021) Loss of activating transcription factor 3 prevents KRAS-mediated pancreatic cancer. Oncogene. 40: 3118-3135
Lazarus J, Oneka MD, Barua S, et al. (2019) Mathematical Modeling of the Metastatic Colorectal Cancer Microenvironment Defines the Importance of Cytotoxic Lymphocyte Infiltration and Presence of PD-L1 on Antigen Presenting Cells. Annals of Surgical Oncology
Wen HJ, Gao S, Wang Y, et al. (2019) Myeloid cell-derived HB-EGF Drives Tissue Recovery After Pancreatitis. Cellular and Molecular Gastroenterology and Hepatology
Wang L, Yang H, Zamperone A, et al. (2019) ATDC is required for the initiation of KRAS-induced pancreatic tumorigenesis. Genes & Development
Pal A, Dziubinski M, Di Magliano MP, et al. (2017) Usp9x Promotes Survival in Human Pancreatic Cancer and Its Inhibition Suppresses Pancreatic Ductal Adenocarcinoma In Vivo Tumor Growth. Neoplasia (New York, N.Y.). 20: 152-164
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