Qingdong Ke, Ph.D.
Affiliations: | 2007 | New York University, New York, NY, United States |
Area:
Toxicology, Environmental Engineering, Molecular BiologyGoogle:
"Qingdong Ke"Cross-listing: E-Tree
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Laulicht F, Brocato J, Ke Q, et al. (2014) Carcinogenicity of Metal Compounds Handbook On the Toxicology of Metals: Fourth Edition. 1: 351-378 |
Li Q, Ke Q, Costa M. (2009) Alterations of histone modifications by cobalt compounds. Carcinogenesis. 30: 1243-51 |
Ke Q, Li Q, Ellen TP, et al. (2008) Nickel compounds induce phosphorylation of histone H3 at serine 10 by activating JNK-MAPK pathway. Carcinogenesis. 29: 1276-81 |
Ke Q, Ellen TP, Costa M. (2008) Nickel compounds induce histone ubiquitination by inhibiting histone deubiquitinating enzyme activity. Toxicology and Applied Pharmacology. 228: 190-9 |
Ellen TP, Ke Q, Zhang P, et al. (2008) NDRG1, a growth and cancer related gene: regulation of gene expression and function in normal and disease states. Carcinogenesis. 29: 2-8 |
Ke Q, Davidson T, Kluz T, et al. (2007) Fluorescent tracking of nickel ions in human cultured cells. Toxicology and Applied Pharmacology. 219: 18-23 |
Ke Q, Costa M. (2006) Hypoxia-inducible factor-1 (HIF-1). Molecular Pharmacology. 70: 1469-80 |
Chen H, Ke Q, Kluz T, et al. (2006) Nickel ions increase histone H3 lysine 9 dimethylation and induce transgene silencing. Molecular and Cellular Biology. 26: 3728-37 |
Ke Q, Davidson T, Chen H, et al. (2006) Alterations of histone modifications and transgene silencing by nickel chloride. Carcinogenesis. 27: 1481-8 |
Ke Q, Li J, Ding J, et al. (2006) Essential role of ROS-mediated NFAT activation in TNF-alpha induction by crystalline silica exposure. American Journal of Physiology. Lung Cellular and Molecular Physiology. 291: L257-64 |