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Matthew W Kay

Affiliations: 
2194-1196 Biomedical Engineering George Washington University, Washington, DC, United States 
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"Matthew Kay"
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Publications

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Stevens JA, Dobratz TC, Fischer KD, et al. (2023) Mechanisms of reduced myocardial energetics of the dystrophic heart. American Journal of Physiology. Heart and Circulatory Physiology
Swift LM, Kay MW, Ripplinger CM, et al. (2021) Stop the beat to see the rhythm: excitation-contraction uncoupling in cardiac research. American Journal of Physiology. Heart and Circulatory Physiology
Moreno A, Kowalik G, Mendelowitz D, et al. (2021) Optogenetic Control of Cardiac Autonomic Neurons in Transgenic Mice. Methods in Molecular Biology (Clifton, N.J.). 2191: 309-321
Entcheva E, Kay MW. (2020) Cardiac optogenetics: a decade of enlightenment. Nature Reviews. Cardiology
Zasadny FM, Dyavanapalli J, Dowling NM, et al. (2020) Cholinergic stimulation improves electrophysiological rate adaptation during pressure overload-induced heart failure in rats. American Journal of Physiology. Heart and Circulatory Physiology
Dyavanapalli J, Rodriguez J, Rocha Dos Santos C, et al. (2020) Activation of Oxytocin Neurons Improves Cardiac Function in a Pressure-Overload Model of Heart Failure. Jacc. Basic to Translational Science. 5: 484-497
Dyavanapalli J, Hora AJ, Escobar JB, et al. (2020) Chemogenetic activation of intracardiac cholinergic neurons improves cardiac function in pressure overload induced heart failure. American Journal of Physiology. Heart and Circulatory Physiology
Lin J, Poelzing S, George SA, et al. (2020) Exploring the Effects of Conduction Reserve and Ephaptic Coupling in Cardiac Cells Biophysical Journal. 118: 494a
Moreno A, Endicott K, Skancke M, et al. (2019) Sudden Heart Rate Reduction Upon Optogenetic Release of Acetylcholine From Cardiac Parasympathetic Neurons in Perfused Hearts. Frontiers in Physiology. 10: 16
Rodriguez Gonzalez J, Dyavanapalli J, Rocha C, et al. (2019) Abstract 307: Chemogenetic Activation of Paraventricular Oxytocin Neurons Reduces Cardiac Dysfunction During Heart Failure Circulation Research. 125
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