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Mark Baker, Ph.D. - Publications

Affiliations: 
Saint Andrews University, Saint Andrews, Scotland, United Kingdom 
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37 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2017 Baker D, Pryce G, Visintin C, Sisay S, Bondarenko AI, Ho WSV, Jackson SJ, Williams TE, Al-Izki S, Sevastou I, Okuyama M, Graier WF, Stevenson LA, Tanner C, Ross R, ... ... Baker MD, et al. Big conductance calcium-activated potassium channel openers control spasticity without sedation. British Journal of Pharmacology. PMID 28677901 DOI: 10.1111/Bph.13889  0.312
2014 Hockley JR, Boundouki G, Cibert-Goton V, McGuire C, Yip PK, Chan C, Tranter M, Wood JN, Nassar MA, Blackshaw LA, Aziz Q, Michael GJ, Baker MD, Winchester WJ, Knowles CH, et al. Multiple roles for NaV1.9 in the activation of visceral afferents by noxious inflammatory, mechanical, and human disease-derived stimuli. Pain. 155: 1962-75. PMID 24972070 DOI: 10.1016/j.pain.2014.06.015  0.442
2013 Lidster K, Jackson SJ, Ahmed Z, Munro P, Coffey P, Giovannoni G, Baker MD, Baker D. Neuroprotection in a novel mouse model of multiple sclerosis. Plos One. 8: e79188. PMID 24223903 DOI: 10.1371/Journal.Pone.0079188  0.324
2013 Hockley JR, Cibert-Goton V, Boundouki G, Wood J, Knowles CH, Baker MD, Winchester W, Bulmer DC. Tu2121 Mechanotransduction in Mouse Visceral Afferent Fibres Is Modulated by Nav1.9 Gastroenterology. 144: S-935. DOI: 10.1016/S0016-5085(13)63476-9  0.353
2012 Eijkelkamp N, Linley JE, Baker MD, Minett MS, Cregg R, Werdehausen R, Rugiero F, Wood JN. Neurological perspectives on voltage-gated sodium channels. Brain : a Journal of Neurology. 135: 2585-612. PMID 22961543 DOI: 10.1093/brain/aws225  0.456
2012 Baker MD, Waxman SG. Sodium channel slow inactivation and adaptation in C-fibres. The Journal of Physiology. 590: 1513-4. PMID 22467918 DOI: 10.1113/jphysiol.2011.224436  0.329
2010 Zhao J, Lee MC, Momin A, Cendan CM, Shepherd ST, Baker MD, Asante C, Bee L, Bethry A, Perkins JR, Nassar MA, Abrahamsen B, Dickenson A, Cobb BS, Merkenschlager M, et al. Small RNAs control sodium channel expression, nociceptor excitability, and pain thresholds. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 30: 10860-71. PMID 20702715 DOI: 10.1523/Jneurosci.1980-10.2010  0.417
2010 Snape A, Pittaway JF, Baker MD. Excitability parameters and sensitivity to anemone toxin ATX-II in rat small diameter primary sensory neurones discriminated by Griffonia simplicifolia isolectin IB4 Journal of Physiology. 588: 125-137. PMID 19900960 DOI: 10.1113/jphysiol.2009.181107  0.324
2009 Shao D, Baker MD, Abrahamsen B, Rugiero F, Malik-Hall M, Poon WY, Cheah KS, Yao KM, Wood JN, Okuse K. A multi PDZ-domain protein Pdzd2 contributes to functional expression of sensory neuron-specific sodium channel Na(V)1.8. Molecular and Cellular Neurosciences. 42: 219-25. PMID 19607921 DOI: 10.1016/j.mcn.2009.07.003  0.474
2008 Ostman JA, Nassar MA, Wood JN, Baker MD. GTP up-regulated persistent Na+ current and enhanced nociceptor excitability require NaV1.9. The Journal of Physiology. 586: 1077-87. PMID 18096591 DOI: 10.1113/jphysiol.2007.147942  0.516
2006 Fertleman CR, Baker MD, Parker KA, Moffatt S, Elmslie FV, Abrahamsen B, Ostman J, Klugbauer N, Wood JN, Gardiner RM, Rees M. SCN9A mutations in paroxysmal extreme pain disorder: allelic variants underlie distinct channel defects and phenotypes. Neuron. 52: 767-74. PMID 17145499 DOI: 10.1016/j.neuron.2006.10.006  0.42
2006 Ekberg J, Jayamanne A, Vaughan CW, Aslan S, Thomas L, Mould J, Drinkwater R, Baker MD, Abrahamsen B, Wood JN, Adams DJ, Christie MJ, Lewis RJ. muO-conotoxin MrVIB selectively blocks Nav1.8 sensory neuron specific sodium channels and chronic pain behavior without motor deficits. Proceedings of the National Academy of Sciences of the United States of America. 103: 17030-5. PMID 17077153 DOI: 10.1073/Pnas.0601819103  0.479
2006 Nassar MA, Baker MD, Levato A, Ingram R, Mallucci G, McMahon SB, Wood JN. Nerve injury induces robust allodynia and ectopic discharges in Nav1.3 null mutant mice. Molecular Pain. 2: 33. PMID 17052333 DOI: 10.1186/1744-8069-2-33  0.42
2006 Foulkes T, Nassar MA, Lane T, Matthews EA, Baker MD, Gerke V, Okuse K, Dickenson AH, Wood JN. Deletion of annexin 2 light chain p11 in nociceptors causes deficits in somatosensory coding and pain behavior. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 26: 10499-507. PMID 17035534 DOI: 10.1523/Jneurosci.1997-06.2006  0.458
2005 Baker MD. Protein kinase C mediates up-regulation of tetrodotoxin-resistant, persistent Na+ current in rat and mouse sensory neurones Journal of Physiology. 567: 851-867. PMID 16002450 DOI: 10.1113/jphysiol.2005.089771  0.311
2005 Stirling LC, Forlani G, Baker MD, Wood JN, Matthews EA, Dickenson AH, Nassar MA. Nociceptor-specific gene deletion using heterozygous NaV1.8-Cre recombinase mice. Pain. 113: 27-36. PMID 15621361 DOI: 10.1016/J.Pain.2004.08.015  0.442
2004 Wood JN, Abrahamsen B, Baker MD, Boorman JD, Donier E, Drew LJ, Nassar MA, Okuse K, Seereeram A, Stirling CL, Zhao J. Ion channel activities implicated in pathological pain. Novartis Foundation Symposium. 261: 32-40; discussion 40. PMID 15469043 DOI: 10.1002/0470869127.Ch4  0.456
2004 Wood JN, Boorman JP, Okuse K, Baker MD. Voltage-gated sodium channels and pain pathways. Journal of Neurobiology. 61: 55-71. PMID 15362153 DOI: 10.1002/neu.20094  0.484
2004 Nassar MA, Stirling LC, Forlani G, Baker MD, Matthews EA, Dickenson AH, Wood JN. Nociceptor-specific gene deletion reveals a major role for Nav1.7 (PN1) in acute and inflammatory pain. Proceedings of the National Academy of Sciences of the United States of America. 101: 12706-11. PMID 15314237 DOI: 10.1073/Pnas.0404915101  0.462
2003 Kiernan MC, Baker MD, Bostock H. Characteristics of late Na(+) current in adult rat small sensory neurons. Neuroscience. 119: 653-60. PMID 12809686 DOI: 10.1016/S0306-4522(03)00194-5  0.662
2003 Baker MD, Chandra SY, Ding Y, Waxman SG, Wood JN. GTP-induced tetrodotoxin-resistant Na+ current regulates excitability in mouse and rat small diameter sensory neurones. The Journal of Physiology. 548: 373-82. PMID 12651922 DOI: 10.1113/jphysiol.2003.039131  0.517
2003 Malik-Hall M, Poon WY, Baker MD, Wood JN, Okuse K. Sensory neuron proteins interact with the intracellular domains of sodium channel NaV1.8. Brain Research. Molecular Brain Research. 110: 298-304. PMID 12591166 DOI: 10.1016/S0169-328X(02)00661-7  0.479
2002 Okuse K, Malik-Hall M, Baker MD, Poon WY, Kong H, Chao MV, Wood JN. Annexin II light chain regulates sensory neuron-specific sodium channel expression. Nature. 417: 653-6. PMID 12050667 DOI: 10.1038/Nature00781  0.47
2002 Wood JN, Akopian AN, Baker M, Ding Y, Geoghegan F, Nassar M, Malik-Hall M, Okuse K, Poon L, Ravenall S, Sukumaran M, Souslova V. Sodium channels in primary sensory neurons: relationship to pain states. Novartis Foundation Symposium. 241: 159-68; discussion 1. PMID 11771644 DOI: 10.1002/0470846682.Ch11  0.541
2001 Wood JN, Baker M. Voltage-gated sodium channels. Current Opinion in Pharmacology. 1: 17-21. PMID 11712529 DOI: 10.1016/S1471-4892(01)00007-8  0.494
2001 Baker MD, Wood JN. Involvement of Na+ channels in pain pathways. Trends in Pharmacological Sciences. 22: 27-31. PMID 11165669 DOI: 10.1016/S0165-6147(00)01585-6  0.495
2000 Baker MD. Selective block of late Na + current by local anaesthetics in rat large sensory neurones British Journal of Pharmacology. 129: 1617-1626. PMID 10780966  0.309
1999 Baker MD, Bostock H. The pH dependence of late sodium current in large sensory neurons. Neuroscience. 92: 1119-30. PMID 10426550 DOI: 10.1016/S0306-4522(99)00058-5  0.643
1998 Baker MD, Bostock H. Inactivation of macroscopic late Na+ current and characteristics of unitary late Na+ currents in sensory neurons. Journal of Neurophysiology. 80: 2538-49. PMID 9819261 DOI: 10.1152/Jn.1998.80.5.2538  0.664
1997 Baker MD, Bostock H. Low-threshold, persistent sodium current in rat large dorsal root ganglion neurons in culture. Journal of Neurophysiology. 77: 1503-13. PMID 9084615 DOI: 10.1152/Jn.1997.77.3.1503  0.659
1992 Baker M, Bostock H. Ectopic activity in demyelinated spinal root axons of the rat. The Journal of Physiology. 451: 539-52. PMID 1403824  0.649
1992 Howe JR, Baker M, Ritchie JM. On the block of outward potassium current in rabbit Schwann cells by internal sodium ions. Proceedings. Biological Sciences / the Royal Society. 249: 309-16. PMID 1359561 DOI: 10.1098/rspb.1992.0120  0.316
1991 Bostock H, Baker M, Grafe P, Reid G. Changes in excitability and accommodation of human motor axons following brief periods of ischaemia. The Journal of Physiology. 441: 513-35. PMID 1816385 DOI: 10.1113/Jphysiol.1991.Sp018765  0.658
1991 Bostock H, Baker M, Reid G. Changes in excitability of human motor axons underlying post-ischaemic fasciculations: evidence for two stable states. The Journal of Physiology. 441: 537-57. PMID 1667800 DOI: 10.1113/Jphysiol.1991.Sp018766  0.67
1989 Baker M, Bostock H. Depolarization changes the mechanism of accommodation in rat and human motor axons. The Journal of Physiology. 411: 545-61. PMID 2614732  0.649
1988 Bostock H, Baker M. Evidence for two types of potassium channel in human motor axons in vivo. Brain Research. 462: 354-8. PMID 3191395 DOI: 10.1016/0006-8993(88)90564-1  0.641
1987 Baker M, Bostock H, Grafe P, Martius P. Function and distribution of three types of rectifying channel in rat spinal root myelinated axons. The Journal of Physiology. 383: 45-67. PMID 2443652 DOI: 10.1113/Jphysiol.1987.Sp016395  0.68
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