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Cheng-Yuan Kao, Ph.D.

Affiliations: 
2005 University of California, Davis, Davis, CA 
Area:
Immunology
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"Cheng-Yuan Kao"
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Reen Wu grad student 2005 UC Davis
 (Molecular characterization of human airway epithelium innate immunity by IL-17 regulation.)

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Chang CS, Liao YC, Huang CT, et al. (2021) Identification of a gut microbiota member that ameliorates DSS-induced colitis in intestinal barrier enhanced Dusp6-deficient mice. Cell Reports. 37: 110016
Kao C, Chang C, Liao Y, et al. (2020) The Novel Roles of Dusp6 in Gut Barrier Modulation and Microbiome Shaping The Faseb Journal. 34: 1-1
Chang CS, Kao CY. (2019) Current understanding of the gut microbiota shaping mechanisms. Journal of Biomedical Science. 26: 59
Hsu WC, Chen MY, Hsu SC, et al. (2018) DUSP6 mediates T cell receptor-engaged glycolysis and restrains T cell differentiation. Proceedings of the National Academy of Sciences of the United States of America
Ruan JW, Statt S, Huang CT, et al. (2016) Dual-specificity phosphatase 6 deficiency regulates gut microbiome and transcriptome response against diet-induced obesity in mice. Nature Microbiology. 2: 16220
Chen HD, Kao CY, Liu BY, et al. (2016) HLH-30/TFEB-mediated autophagy functions in a cell-autonomous manner for epithelium intrinsic cellular defense against bacterial pore-forming toxin in C. elegans. Autophagy. 0
Statt S, Ruan JW, Huang CT, et al. (2015) Lipidome and transcriptome profiling of pneumolysin intoxication identifies networks involved in statin-conferred protection of airway epithelial cells. Scientific Reports. 5: 10624
Statt S, Ruan JW, Hung LY, et al. (2015) Statins Enhance Cellular Resistance Against Bacterial Pore-forming Toxins in Airway Epithelial Cells. American Journal of Respiratory Cell and Molecular Biology
Kao CY, Los FC, Huffman DL, et al. (2011) Global functional analyses of cellular responses to pore-forming toxins. Plos Pathogens. 7: e1001314
Los FC, Kao CY, Smitham J, et al. (2011) RAB-5- and RAB-11-dependent vesicle-trafficking pathways are required for plasma membrane repair after attack by bacterial pore-forming toxin. Cell Host & Microbe. 9: 147-57
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