Ri-Xin Zhang
Affiliations: | National Institute for Dental and Craniofacial Research, Bethesda, MD, United States |
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"Ri-Xin Zhang"Mean distance: 17.29 (cluster 31) | S | N | B | C | P |
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| Zhang RX, Ren K, Dubner R. (2013) Osteoarthritis pain mechanisms: basic studies in animal models. Osteoarthritis and Cartilage / Oars, Osteoarthritis Research Society. 21: 1308-15 |
| Li A, Zhang Y, Lao L, et al. (2011) Serotonin Receptor 2A/C Is Involved in Electroacupuncture Inhibition of Pain in an Osteoarthritis Rat Model. Evidence-Based Complementary and Alternative Medicine : Ecam. 2011: 619650 |
| Li A, Wang Y, Xin J, et al. (2007) Electroacupuncture suppresses hyperalgesia and spinal Fos expression by activating the descending inhibitory system. Brain Research. 1186: 171-9 |
| Zhang RX, Liu B, Qiao JT, et al. (2005) Electroacupuncture suppresses spinal expression of neurokinin-1 receptors induced by persistent inflammation in rats. Neuroscience Letters. 384: 339-43 |
| Zhang RX, Liu B, Lao L, et al. (2005) Spinal preprodynorphin mRNA expression in neonatal rats following peripheral inflammation. Brain Research. 1038: 238-42 |
| Zhang RX, Lao L, Qiao JT, et al. (2004) Endogenous and exogenous glucocorticoid suppresses up-regulation of preprodynorphin mRNA and hyperalgesia in rats with peripheral inflammation. Neuroscience Letters. 359: 85-8 |
| Zhang RX, Lao L, Qiao JT, et al. (2004) Effects of aging on hyperalgesia and spinal dynorphin expression in rats with peripheral inflammation. Brain Research. 999: 135-41 |
| Zhang RX, Lao L, Qiao JT, et al. (2003) Strain differences in pain sensitivity and expression of preprodynorphin mRNA in rats following peripheral inflammation. Neuroscience Letters. 353: 213-6 |
| Zhang RX, Wang H, Ruda M, et al. (1998) c-Fos expression in NMDA receptor-contained neurons in spinal cord in a rat model of inflammation: a double immunocytochemical study. Brain Research. 795: 282-6 |
| Zhang RX, Ruda MA, Qiao JT. (1998) Pre-emptive intrathecal Mk-801, a non-competitive N-methyl-D-aspartate receptor antagonist, inhibits the up-regulation of spinal dynorphin mRNA and hyperalgesia in a rat model of chronic inflammation. Neuroscience Letters. 241: 57-60 |