Emily Bailey, Ph.D.
Affiliations: | 2013 | Cellular and Molecualr Medicine | Johns Hopkins University, Baltimore, MD |
Area:
Oncology, Molecular BiologyGoogle:
"Emily Bailey"Parents
Sign in to add mentor| David Husu | grad student | 2013 | Johns Hopkins | |
| (Generation of a FLT3 kinase domain point mutation (D835Y) knock-in mouse confers less aggressive disease than mice with a FLT3/ITD.) | ||||
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| Bailey E, Li L, Duffield AS, et al. (2013) FLT3/D835Y mutation knock-in mice display less aggressive disease compared with FLT3/internal tandem duplication (ITD) mice. Proceedings of the National Academy of Sciences of the United States of America. 110: 21113-8 |
| Bailey EJ, Duffield AS, Greenblatt SM, et al. (2013) Effect of FLT3 ligand on survival and disease phenotype in murine models harboring a FLT3 internal tandem duplication mutation. Comparative Medicine. 63: 218-26 |
| Li L, Bailey E, Greenblatt S, et al. (2011) Loss of the wild-type allele contributes to myeloid expansion and disease aggressiveness in FLT3/ITD knockin mice. Blood. 118: 4935-45 |
| Zheng R, Bailey E, Nguyen B, et al. (2011) Further activation of FLT3 mutants by FLT3 ligand. Oncogene. 30: 4004-14 |
| Bailey E, Li L, Huso D, et al. (2011) Generation of a FLT3 Kinase Domain Point Mutation (D835Y equivalent) Knock-in Mouse Confers Less Aggressive Disease Compared with FLT3 Internal Tandem Duplication (ITD) Mice Blood. 118: 924-924 |