Sebastian Bonhoeffer

ETH/Uni Zurich, Zürich, Zürich, Switzerland 
"Sebastian Bonhoeffer"


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Robert M. May grad student Oxford
Martin Nowak grad student


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Lei Sun grad student ETH Zürich
Ruy M. Ribeiro grad student 1996-1999 Oxford
Martin Ackermann post-doc ETH Zürich
Orkun  S. Soyer post-doc 2004-2006 ETH Zürich (Chemistry Tree)
Dusan Misevic post-doc 2006-2009 ETH/Uni Zurich
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Vasse M, Bonhoeffer S, Frenoy A. (2022) Ecological effects of stress drive bacterial evolvability under sub-inhibitory antibiotic treatments. Isme Communications. 2: 80
Angst DC, Tepekule B, Sun L, et al. (2021) Comparing treatment strategies to reduce antibiotic resistance in an in vitro epidemiological setting. Proceedings of the National Academy of Sciences of the United States of America. 118
Benz F, Huisman JS, Bakkeren E, et al. (2020) Plasmid- and strain-specific factors drive variation in ESBL-plasmid spread in vitro and in vivo. The Isme Journal
Tepekule B, Abel Zur Wiesch P, Kouyos RD, et al. (2019) Quantifying the impact of treatment history on plasmid-mediated resistance evolution in human gut microbiota. Proceedings of the National Academy of Sciences of the United States of America
Sun L, Ashcroft P, Ackermann M, et al. (2019) Stochastic gene expression influences the selection of antibiotic resistance mutations. Molecular Biology and Evolution
Bakkeren E, Huisman JS, Fattinger SA, et al. (2019) Salmonella persisters promote the spread of antibiotic resistance plasmids in the gut. Nature. 573: 276-280
Frenoy A, Bonhoeffer S. (2018) Death and population dynamics affect mutation rate estimates and evolvability under stress in bacteria. Plos Biology. 16: e2005056
Hozé N, Bonhoeffer S, Regoes R. (2018) Assessing the public health impact of tolerance-based therapies with mathematical models. Plos Computational Biology. 14: e1006119
Sun L, Alexander HK, Bogos B, et al. (2018) Effective polyploidy causes phenotypic delay and influences bacterial evolvability. Plos Biology. 16: e2004644
Kühnert D, Kouyos R, Shirreff G, et al. (2018) Quantifying the fitness cost of HIV-1 drug resistance mutations through phylodynamics. Plos Pathogens. 14: e1006895
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