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Matthew T. Lewis - Publications

Affiliations: 
Physiology Michigan State University, East Lansing, MI 
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11 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2022 Lewis MT, Levitsky Y, Bazil JN, Wiseman RW. Measuring Mitochondrial Function: From Organelle to Organism. Methods in Molecular Biology (Clifton, N.J.). 2497: 141-172. PMID 35771441 DOI: 10.1007/978-1-0716-2309-1_10  0.556
2021 Lewis MT, Blain GM, Hart CR, Layec G, Rossman MJ, Park SY, Trinity JD, Gifford JR, Sidhu SK, Weavil JC, Hureau TJ, Jessop JE, Bledsoe AD, Amann M, Richardson RS. Acute high-intensity exercise and skeletal muscle mitochondrial respiratory function: The role of metabolic perturbation. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. PMID 34549627 DOI: 10.1152/ajpregu.00158.2021  0.348
2020 Frisbee JC, Halvorson BD, Lewis MT, Wiseman RW. Shifted vascular optimization: The emergence of a new arteriolar behaviour with chronic metabolic disease. Experimental Physiology. PMID 32045062 DOI: 10.1113/Ep087871  0.615
2019 Lewis MT, Kasper JD, Bazil JN, Frisbee JC, Wiseman RW. Quantification of Mitochondrial Oxidative Phosphorylation in Metabolic Disease: Application to Type 2 Diabetes. International Journal of Molecular Sciences. 20. PMID 31652915 DOI: 10.3390/Ijms20215271  0.681
2019 Lewis MT, Lujan HL, Wiseman RW, DiCarlo SE. The hypertension advantage and natural selection: Since type 2 diabetes associates with co-morbidities and premature death, why have the genetic variants remained in the human genome? Medical Hypotheses. 129: 109237. PMID 31371084 DOI: 10.1016/J.Mehy.2019.109237  0.542
2019 Lewis MT, Kasper JD, Bazil JN, Frisbee JC, Wiseman RW. Skeletal Muscle Energetics are Compromised Only During High Intensity Contractions in the Goto-Kakizaki Rat Model of Type 2 Diabetes. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. PMID 31188651 DOI: 10.1152/Ajpregu.00127.2019  0.672
2019 Diaz-Otero JM, Yen TC, Ahmad A, Laimon-Thomson E, Abolibdeh B, Kelly K, Lewis MT, Wiseman RW, Jackson WF, Dorrance AM. Transient Receptor Potential Vanilloid 4 Channels are Important Regulators of Parenchymal Arteriole Dilation and Cognitive Function. Microcirculation (New York, N.Y. : 1994). e12535. PMID 30721555 DOI: 10.1111/Micc.12535  0.496
2019 Lewis MT, Lujan HL, Tonson A, Wiseman RW, DiCarlo SE. Obesity and inactivity, not hyperglycemia, cause exercise intolerance in individuals with type 2 diabetes: Solving the obesity and inactivity versus hyperglycemia causality dilemma. Medical Hypotheses. 123: 110-114. PMID 30696579 DOI: 10.1016/J.Mehy.2019.01.013  0.549
2018 Frisbee JC, Lewis MT, Kasper JD, Chantler PD, Wiseman RW. TYPE II DIABETES MELLITUS IN THE GOTO-KAKIZAKI RAT IMPAIRS MICROVASCULAR FUNCTION AND CONTRIBUTES TO PREMATURE SKELETAL MUSCLE FATIGUE. Journal of Applied Physiology (Bethesda, Md. : 1985). PMID 30571284 DOI: 10.1152/Japplphysiol.00751.2018  0.679
2018 Frisbee JC, Lewis MT, Wiseman RW. Skeletal muscle performance in metabolic disease: microvascular or mitochondrial limitation or both? Microcirculation (New York, N.Y. : 1994). e12517. PMID 30471168 DOI: 10.1111/Micc.12517  0.651
2018 Lemaster KA, Frisbee SJ, DuBois L, Tzemos N, Wu F, Lewis MT, Wiseman RW, Frisbee JC. CHRONIC ATORVASTATIN AND EXERCISE CAN PARTIALLY REVERSE ESTABLISHED SKELETAL MUSCLE MICROVASCULOPATHY IN METABOLIC SYNDROME. American Journal of Physiology. Heart and Circulatory Physiology. PMID 29932769 DOI: 10.1152/Ajpheart.00193.2018  0.634
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