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| Liu X, Zheng H, Li X, et al. (2016) Gain-of-function mutations of Ptpn11 (Shp2) cause aberrant mitosis and increase susceptibility to DNA damage-induced malignancies. Proceedings of the National Academy of Sciences of the United States of America |
| Cordeddu V, Yin JC, Gunnarsson C, et al. (2015) Activating mutations affecting the Dbl homology domain of SOS2 cause Noonan syndrome. Human Mutation |
| St-Germain JR, Taylor P, Zhang W, et al. (2015) Differential regulation of FGFR3 by PTPN1 and PTPN2. Proteomics. 15: 419-33 |
| Goodwin CB, Li XJ, Mali RS, et al. (2014) PI3K p110δ uniquely promotes gain-of-function Shp2-induced GM-CSF hypersensitivity in a model of JMML. Blood. 123: 2838-42 |
| Yang W, Wang J, Moore DC, et al. (2013) Ptpn11 deletion in a novel progenitor causes metachondromatosis by inducing hedgehog signalling. Nature. 499: 491-5 |
| Abram CL, Roberge GL, Pao LI, et al. (2013) Distinct roles for neutrophils and dendritic cells in inflammation and autoimmunity in motheaten mice. Immunity. 38: 489-501 |
| Mori J, Wang YJ, Ellison S, et al. (2012) Dominant role of the protein-tyrosine phosphatase CD148 in regulating platelet activation relative to protein-tyrosine phosphatase-1B. Arteriosclerosis, Thrombosis, and Vascular Biology. 32: 2956-65 |
| Yang Z, Xue B, Umitsu M, et al. (2012) The signaling adaptor GAB1 regulates cell polarity by acting as a PAR protein scaffold. Molecular Cell. 47: 469-83 |
| Wu X, Yin J, Simpson J, et al. (2012) Increased BRAF heterodimerization is the common pathogenic mechanism for noonan syndrome-associated RAF1 mutants. Molecular and Cellular Biology. 32: 3872-90 |
| Haj FG, Sabet O, Kinkhabwala A, et al. (2012) Regulation of signaling at regions of cell-cell contact by endoplasmic reticulum-bound protein-tyrosine phosphatase 1B. Plos One. 7: e36633 |