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Singh DR, Nelson SE, Pawelski AS, et al. (2023) Epstein-Barr virus LMP1 protein promotes proliferation and inhibits differentiation of epithelial cells via activation of YAP and TAZ. Proceedings of the National Academy of Sciences of the United States of America. 120: e2219755120 |
Ali A, Ohashi M, Casco A, et al. (2022) Rta is the principal activator of Epstein-Barr virus epithelial lytic transcription. Plos Pathogens. 18: e1010886 |
Damania B, Kenney SC, Raab-Traub N. (2022) Epstein-Barr virus: Biology and clinical disease. Cell. 185: 3652-3670 |
Van Sciver N, Ohashi M, Nawandar DM, et al. (2021) ΔNp63α promotes Epstein-Barr virus latency in undifferentiated epithelial cells. Plos Pathogens. 17: e1010045 |
Van Sciver N, Ohashi M, Pauly NP, et al. (2021) Hippo signaling effectors YAP and TAZ induce Epstein-Barr Virus (EBV) lytic reactivation through TEADs in epithelial cells. Plos Pathogens. 17: e1009783 |
Li C, Romero-Masters JC, Huebner S, et al. (2020) EBNA2-deleted Epstein-Barr virus (EBV) isolate, P3HR1, causes Hodgkin-like lymphomas and diffuse large B cell lymphomas with type II and Wp-restricted latency types in humanized mice. Plos Pathogens. 16: e1008590 |
Romero-Masters JC, Ohashi M, Djavadian R, et al. (2020) An EBNA3A-mutated Epstein-Barr virus (EBV) retains the capacity for lymphomagenesis in a cord blood-humanized mouse model. Journal of Virology |
Romero-Masters JC, Huebner SM, Ohashi M, et al. (2020) B cells infected with Type 2 Epstein-Barr virus (EBV) have increased NFATc1/NFATc2 activity and enhanced lytic gene expression in comparison to Type 1 EBV infection. Plos Pathogens. 16: e1008365 |
Eichelberg MR, Welch R, Guidry JT, et al. (2019) Epstein-Barr Virus Infection Promotes Epithelial Cell Growth by Attenuating Differentiation-Dependent Exit from the Cell Cycle. Mbio. 10 |
Romero-Masters JC, Ohashi M, Djavadian R, et al. (2018) An EBNA3C-deleted Epstein-Barr virus (EBV) mutant causes B-cell lymphomas with delayed onset in a cord blood-humanized mouse model. Plos Pathogens. 14: e1007221 |