Carl W. Tong, Ph.D.
Affiliations: | 2002 | The Texas A&M University System Health Science Center |
Area:
Animal Physiology Biology, Medical Biophysics, BiochemistryGoogle:
"Carl Tong"Mean distance: (not calculated yet)
Parents
Sign in to add mentor| Mariappan Muthuchamy | grad student | 2002 | The Texas A&M University System Health Science Center | |
| (Molecular mechanisms of cardiac contractility: Role of myofilament proteins.) | ||||
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Publications
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| Rosas PC, Warren CM, Creed HA, et al. (2019) Cardiac Myosin Binding Protein-C Phosphorylation Mitigates Age-Related Cardiac Dysfunction: Hope for Better Aging? Jacc. Basic to Translational Science. 4: 817-830 |
| Rosas PC, Warren CM, Batra A, et al. (2019) Abstract 621: Cardiac Myosin Binding Protein-C Phosphorylation in Ser-273 and Ser-282 is Critical to Maintain Cardiac Function During Aging Circulation Research. 125 |
| Liu Y, Wang J, Li J, et al. (2017) Deletion of Cdc42 in embryonic cardiomyocytes results in right ventricle hypoplasia. Clinical and Translational Medicine. 6: 40 |
| Tong CW, Madhur MS, Rzeszut AK, et al. (2017) Status of Early-Career Academic Cardiology: A Global Perspective. Journal of the American College of Cardiology. 70: 2290-2303 |
| Tong CW, Dusio GF, Govindan S, et al. (2017) Usefulness of Released Cardiac Myosin Binding Protein-C as a Predictor of Cardiovascular Events. The American Journal of Cardiology |
| Tong CW, Sharma G, Rzeszut A, et al. (2017) ACUTE DECOMPENSATED HEART FAILURE: THE JOURNEY FROM PATIENTS’ PERSPECTIVE Journal of the American College of Cardiology. 69: 789 |
| Tong CW, Rosas P, Kidwell D, et al. (2017) CARDIAC MYOSIN BINDING PROTEIN-C DE-PHOSPHORYLATION MEDIATED SLOWING OF CROSS-DETACHMENT WORSENS HEART FAILURE Journal of the American College of Cardiology. 69: 670 |
| Dusio G, Liu Y, Rosas P, et al. (2016) ABSENCE OF CARDIAC MYOSIN BINDING PROTEIN-C INCREASES INFLAMMATORY DAMAGE TO THE HEART Journal of the American College of Cardiology. 67: 1396 |
| Rosas PC, Liu Y, Abdalla MI, et al. (2015) Phosphorylation of cardiac Myosin-binding protein-C is a critical mediator of diastolic function. Circulation. Heart Failure. 8: 582-94 |
| Tong CW, Wu X, Liu Y, et al. (2015) Phosphoregulation of Cardiac Inotropy via Myosin Binding Protein-C During Increased Pacing Frequency or β1-Adrenergic Stimulation. Circulation. Heart Failure. 8: 595-604 |