Richard R. Kew
Affiliations: | Stony Brook University, Stony Brook, NY, United States |
Area:
Immunology, Molecular Biology, Cell BiologyGoogle:
"Richard Kew"Children
Sign in to add trainee| Glenda Trujillo | grad student | 2004 | SUNY Stony Brook |
| Lauren A. McVoy | grad student | 2005 | SUNY Stony Brook |
| David M. Habiel | grad student | 2012 | SUNY Stony Brook |
| Lingyin Ge | grad student | 2013 | SUNY Stony Brook |
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Publications
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| Kew RR. (2019) The Vitamin D Binding Protein and Inflammatory Injury: A Mediator or Sentinel of Tissue Damage? Frontiers in Endocrinology. 10: 470 |
| Kew RR, Tabrizian T, Vosswinkel JA, et al. (2018) Vitamin D Binding Protein (DBP) Deficiency in Mice Decreases Systemic and Select Tissue Levels of Inflammatory Cytokines in a Murine Model of Acute Muscle Injury. The Journal of Trauma and Acute Care Surgery |
| Espaillat MP, Snider AJ, Qiu Z, et al. (2017) Loss of acid ceramidase in myeloid cells suppresses intestinal neutrophil recruitment. Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology |
| Espaillat MP, Kew RR, Obeid LM. (2016) Sphingolipids in neutrophil function and inflammatory responses: Mechanisms and implications for intestinal immunity and inflammation in ulcerative colitis. Advances in Biological Regulation |
| Pednekar L, Valentino A, Ji Y, et al. (2016) Identification of the gC1qR sites for the HIV-1 viral envelope protein gp41 and the HCV core protein: Implications in viral-specific pathogenesis and therapy. Molecular Immunology. 74: 18-26 |
| Ramadass M, Ghebrehiwet B, Kew RR. (2015) Enhanced recognition of plasma proteins in a non-native state by complement C3b. A possible clearance mechanism for damaged proteins in blood. Molecular Immunology. 64: 55-62 |
| Penzo M, Habiel DM, Ramadass M, et al. (2014) Cell migration to CXCL12 requires simultaneous IKKα and IKKβ-dependent NF-κB signaling. Biochimica Et Biophysica Acta. 1843: 1796-804 |
| Ramadass M, Ghebrehiwet B, Smith RJ, et al. (2014) Generation of multiple fluid-phase C3b:plasma protein complexes during complement activation: possible implications in C3 glomerulopathies. Journal of Immunology (Baltimore, Md. : 1950). 192: 1220-30 |
| Ghebrehiwet B, Ji Y, Valentino A, et al. (2014) Soluble gC1qR is an autocrine signal that induces B1R expression on endothelial cells. Journal of Immunology (Baltimore, Md. : 1950). 192: 377-84 |
| Ge L, Trujillo G, Miller EJ, et al. (2014) Circulating complexes of the vitamin D binding protein with G-actin induce lung inflammation by targeting endothelial cells. Immunobiology. 219: 198-207 |