Paul Bornstein, M.D.

Affiliations: 
2007 Biochemistry University of Washington, Seattle, Seattle, WA 
Area:
collagen structure and synthesis, matricellular protein biochemistry
Website:
http://www.sfbmec.fr/?p=1485
Google:
"Paul Bornstein"
Bio:

(1934 - 2013)

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Publications

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Huang Z, Newcomb CJ, Lei Y, et al. (2015) Bioactive nanofibers enable the identification of thrombospondin 2 as a key player in enamel regeneration. Biomaterials. 61: 216-28
Schultz GS, Davidson JM, Kirsner RS, et al. (2011) Dynamic reciprocity in the wound microenvironment. Wound Repair and Regeneration : Official Publication of the Wound Healing Society [and] the European Tissue Repair Society. 19: 134-48
Frolova EG, Pluskota E, Krukovets I, et al. (2010) Thrombospondin-4 regulates vascular inflammation and atherogenesis. Circulation Research. 107: 1313-25
Card L, Henderson N, Zhang Y, et al. (2010) Expression in SPARC-null mice of collagen type I lacking the globular domain of the α1(I) N-propeptide results in abdominal hernias and loss of dermal collagen. Matrix Biology : Journal of the International Society For Matrix Biology. 29: 559-64
Bornstein P. (2010) Collagen: relatively invariant (helical) and variable (nonhelical) regions. Science (New York, N.Y.). 161: 592-3
Bornstein P. (2009) Thrombospondins function as regulators of angiogenesis. Journal of Cell Communication and Signaling. 3: 189-200
Bornstein P. (2009) Matricellular proteins: an overview. Journal of Cell Communication and Signaling. 3: 163-5
Kyriakides TR, Wulsin D, Skokos EA, et al. (2009) Mice that lack matrix metalloproteinase-9 display delayed wound healing associated with delayed reepithelization and disordered collagen fibrillogenesis. Matrix Biology : Journal of the International Society For Matrix Biology. 28: 65-73
MacLauchlan S, Skokos EA, Meznarich N, et al. (2009) Macrophage fusion, giant cell formation, and the foreign body response require matrix metalloproteinase 9. Journal of Leukocyte Biology. 85: 617-26
Maclauchlan S, Skokos EA, Agah A, et al. (2009) Enhanced angiogenesis and reduced contraction in thrombospondin-2-null wounds is associated with increased levels of matrix metalloproteinases-2 and -9, and soluble VEGF. The Journal of Histochemistry and Cytochemistry : Official Journal of the Histochemistry Society. 57: 301-13
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