Jose V. Llorens

Affiliations: 
Uppsala University, Uppsala, Uppsala län, Sweden 
Area:
Friedreich ataxia, mitochondria, neurodegeneration, oxidative stress
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"Jose Llorens"
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Llorens JV, Soriano S, Calap-Quintana P, et al. (2019) The Role of Iron in Friedreich's Ataxia: Insights From Studies in Human Tissues and Cellular and Animal Models. Frontiers in Neuroscience. 13: 75
Monnier V, Llorens JV, Navarro JA. (2018) Impact of Models in the Study and Treatment of Friedreich's Ataxia. International Journal of Molecular Sciences. 19
Calap-Quintana P, Navarro JA, González-Fernández J, et al. (2018) Models of Friedreich's Ataxia. Biomed Research International. 2018: 5065190
Calap-Quintana P, González-Fernández J, Sebastiá-Ortega N, et al. (2017) Drosophila melanogaster Models of Metal-Related Human Diseases and Metal Toxicity International Journal of Molecular Sciences. 18: 1456
Soriano S, Calap-Quintana P, Llorens JV, et al. (2016) Metal Homeostasis Regulators Suppress FRDA Phenotypes in a Drosophila Model of the Disease. Plos One. 11: e0159209
Llorens JV, Metzendorf C, Missirlis F, et al. (2015) Mitochondrial iron supply is required for the developmental pulse of ecdysone biosynthesis that initiates metamorphosis in Drosophila melanogaster. Journal of Biological Inorganic Chemistry : Jbic : a Publication of the Society of Biological Inorganic Chemistry. 20: 1229-38
Calap-Quintana P, Soriano S, Llorens JV, et al. (2015) TORC1 Inhibition by Rapamycin Promotes Antioxidant Defences in a Drosophila Model of Friedreich's Ataxia. Plos One. 10: e0132376
Llorens JV, Metzendorf C, Missirlis F, et al. (2015) Mitochondrial iron supply is required for the developmental pulse of ecdysone biosynthesis that initiates metamorphosis in Drosophila melanogaster Journal of Biological Inorganic Chemistry. 20: 1229-1238
Soriano S, Llorens JV, Blanco-Sobero L, et al. (2013) Deferiprone and idebenone rescue frataxin depletion phenotypes in a Drosophila model of Friedreich's ataxia. Gene. 521: 274-81
Navarro JA, Llorens JV, Soriano S, et al. (2011) Overexpression of human and fly frataxins in Drosophila provokes deleterious effects at biochemical, physiological and developmental levels. Plos One. 6: e21017
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