Daniel P. Kane, Ph.D. - Publications

Affiliations: 
Biology Tufts University, Boston 
Area:
DNA repair

7 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2019 Xing X, Kane DP, Bulock CR, Moore EA, Sharma S, Chabes A, Shcherbakova PV. A recurrent cancer-associated substitution in DNA polymerase ε produces a hyperactive enzyme. Nature Communications. 10: 374. PMID 30670691 DOI: 10.1038/S41467-018-08145-2  0.465
2018 Barbari SR, Kane DP, Moore EA, Shcherbakova PV. Functional Analysis of Cancer-Associated DNA Polymerase ε Variants in Saccharomyces cerevisiae. G3 (Bethesda, Md.). PMID 29352080 DOI: 10.1534/G3.118.200042  0.472
2014 Kane DP, Shcherbakova PV. A common cancer-associated DNA polymerase ε mutation causes an exceptionally strong mutator phenotype, indicating fidelity defects distinct from loss of proofreading. Cancer Research. 74: 1895-901. PMID 24525744 DOI: 10.1158/0008-5472.Can-13-2892  0.464
2012 Kane DP, Shusterman M, Rong Y, McVey M. Competition between replicative and translesion polymerases during homologous recombination repair in Drosophila. Plos Genetics. 8: e1002659. PMID 22532806 DOI: 10.1371/Journal.Pgen.1002659  0.511
2010 Witsell A, Kane DP, McVey M. Super-sized deletions: improved transposon excision screens using a mus309 mutant background. Fly. 4: 137-40. PMID 20081362 DOI: 10.4161/Fly.4.2.10918  0.56
2009 Witsell A, Kane DP, Rubin S, McVey M. Removal of the bloom syndrome DNA helicase extends the utility of imprecise transposon excision for making null mutations in Drosophila. Genetics. 183: 1187-93. PMID 19687136 DOI: 10.1534/Genetics.109.108472  0.548
2007 Conrad LJ, Bai L, Ahern K, Dusinberre K, Kane DP, Brutnell TP. State II dissociation element formation following activator excision in maize. Genetics. 177: 737-47. PMID 17720925 DOI: 10.1534/Genetics.107.075770  0.435
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