Daniel P. Kane, Ph.D. - Publications
Affiliations: | Biology | Tufts University, Boston |
Area:
DNA repairYear | Citation | Score | |||
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2019 | Xing X, Kane DP, Bulock CR, Moore EA, Sharma S, Chabes A, Shcherbakova PV. A recurrent cancer-associated substitution in DNA polymerase ε produces a hyperactive enzyme. Nature Communications. 10: 374. PMID 30670691 DOI: 10.1038/S41467-018-08145-2 | 0.465 | |||
2018 | Barbari SR, Kane DP, Moore EA, Shcherbakova PV. Functional Analysis of Cancer-Associated DNA Polymerase ε Variants in Saccharomyces cerevisiae. G3 (Bethesda, Md.). PMID 29352080 DOI: 10.1534/G3.118.200042 | 0.472 | |||
2014 | Kane DP, Shcherbakova PV. A common cancer-associated DNA polymerase ε mutation causes an exceptionally strong mutator phenotype, indicating fidelity defects distinct from loss of proofreading. Cancer Research. 74: 1895-901. PMID 24525744 DOI: 10.1158/0008-5472.Can-13-2892 | 0.464 | |||
2012 | Kane DP, Shusterman M, Rong Y, McVey M. Competition between replicative and translesion polymerases during homologous recombination repair in Drosophila. Plos Genetics. 8: e1002659. PMID 22532806 DOI: 10.1371/Journal.Pgen.1002659 | 0.511 | |||
2010 | Witsell A, Kane DP, McVey M. Super-sized deletions: improved transposon excision screens using a mus309 mutant background. Fly. 4: 137-40. PMID 20081362 DOI: 10.4161/Fly.4.2.10918 | 0.56 | |||
2009 | Witsell A, Kane DP, Rubin S, McVey M. Removal of the bloom syndrome DNA helicase extends the utility of imprecise transposon excision for making null mutations in Drosophila. Genetics. 183: 1187-93. PMID 19687136 DOI: 10.1534/Genetics.109.108472 | 0.548 | |||
2007 | Conrad LJ, Bai L, Ahern K, Dusinberre K, Kane DP, Brutnell TP. State II dissociation element formation following activator excision in maize. Genetics. 177: 737-47. PMID 17720925 DOI: 10.1534/Genetics.107.075770 | 0.435 | |||
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