| Year |
Citation |
Score |
| 2022 |
Guo D, Daman K, Chen JJ, Shi MJ, Yan J, Matijasevic Z, Rickard AM, Bennett MH, Kiselyov A, Zhou H, Bang AG, Wagner KR, Maehr R, King OD, Hayward LJ, et al. iMyoblasts for ex vivo and in vivo investigations of human myogenesis and disease modeling. Elife. 11. PMID 35076017 DOI: 10.7554/eLife.70341 |
0.319 |
|
| 2015 |
Khogali S, Lucas B, Ammar T, Dejong D, Barbalinardo M, Hayward LJ, Renaud JM. Physiological basis for muscle stiffness and weakness in a knock-in M1592V mouse model of hyperkalemic periodic paralysis. Physiological Reports. 3. PMID 26702073 DOI: 10.14814/phy2.12656 |
0.483 |
|
| 2015 |
Ammar T, Lin W, Higgins A, Hayward LJ, Renaud JM. Understanding the physiology of the asymptomatic diaphragm of the M1592V hyperkalemic periodic paralysis mouse. The Journal of General Physiology. 146: 509-25. PMID 26621775 DOI: 10.1085/jgp.201511476 |
0.367 |
|
| 2014 |
Lucas B, Ammar T, Khogali S, DeJong D, Barbalinardo M, Nishi C, Hayward LJ, Renaud JM. Contractile abnormalities of mouse muscles expressing hyperkalemic periodic paralysis mutant NaV1.4 channels do not correlate with Na+ influx or channel content. Physiological Genomics. 46: 385-97. PMID 24714718 DOI: 10.1152/physiolgenomics.00166.2013 |
0.507 |
|
| 2012 |
Renaud JM, Hayward LJ. Lessons learned from muscle fatigue: implications for treatment of patients with hyperkalemic periodic paralysis. Recent Patents On Biotechnology. 6: 184-91. PMID 23092434 DOI: 10.2174/1872208311206030184 |
0.371 |
|
| 2011 |
Clausen T, Nielsen OB, Clausen JD, Pedersen TH, Hayward LJ. Na+,K+-pump stimulation improves contractility in isolated muscles of mice with hyperkalemic periodic paralysis. The Journal of General Physiology. 138: 117-30. PMID 21708955 DOI: 10.1085/jgp.201010586 |
0.45 |
|
| 2008 |
Hayward LJ, Kim JS, Lee MY, Zhou H, Kim JW, Misra K, Salajegheh M, Wu FF, Matsuda C, Reid V, Cros D, Hoffman EP, Renaud JM, Cannon SC, Brown RH. Targeted mutation of mouse skeletal muscle sodium channel produces myotonia and potassium-sensitive weakness. The Journal of Clinical Investigation. 118: 1437-49. PMID 18317596 DOI: 10.1172/Jci32638 |
0.608 |
|
| 1999 |
Hayward LJ, Sandoval GM, Cannon SC. Defective slow inactivation of sodium channels contributes to familial periodic paralysis. Neurology. 52: 1447-53. PMID 10227633 DOI: 10.1212/Wnl.52.7.1447 |
0.645 |
|
| 1998 |
Andreu AL, Bruno C, Shanske S, Shtilbans A, Hirano M, Krishna S, Hayward L, Systrom DS, Brown RH, DiMauro S. Missense mutation in the mtDNA cytochrome b gene in a patient with myopathy. Neurology. 51: 1444-7. PMID 9818877 DOI: 10.1212/Wnl.51.5.1444 |
0.483 |
|
| 1997 |
Green DS, Hayward LJ, George AL, Cannon SC. A proposed mutation, Val781Ile, associated with hyperkalemic periodic paralysis and cardiac dysrhythmia is a benign polymorphism. Annals of Neurology. 42: 253-6. PMID 9266738 DOI: 10.1002/Ana.410420219 |
0.605 |
|
| 1997 |
Hayward LJ, Brown RH, Cannon SC. Slow inactivation differs among mutant Na channels associated with myotonia and periodic paralysis. Biophysical Journal. 72: 1204-19. PMID 9138567 DOI: 10.1016/S0006-3495(97)78768-X |
0.687 |
|
| 1996 |
Hayward LJ, Brown RH, Cannon SC. Inactivation defects caused by myotonia-associated mutations in the sodium channel III-IV linker. The Journal of General Physiology. 107: 559-76. PMID 8740371 DOI: 10.1085/Jgp.107.5.559 |
0.659 |
|
| 1995 |
Cannon SC, Hayward LJ, Beech J, Brown RH. Sodium channel inactivation is impaired in equine hyperkalemic periodic paralysis. Journal of Neurophysiology. 73: 1892-9. PMID 7623088 DOI: 10.1152/Jn.1995.73.5.1892 |
0.661 |
|
| 1991 |
Hayward L, Wesselmann U, Rymer WZ. Effects of muscle fatigue on mechanically sensitive afferents of slow conduction velocity in the cat triceps surae. Journal of Neurophysiology. 65: 360-70. PMID 2016646 DOI: 10.1152/Jn.1991.65.2.360 |
0.325 |
|
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