Miriam H. Meisler
Affiliations: | University of Michigan, Ann Arbor, Ann Arbor, MI |
Area:
Genetics, Neuroscience BiologyGoogle:
"Miriam Meisler"Cross-listing: Neurotree
Children
Sign in to add traineeNicholas W. Plummer | grad student | 1998 | University of Michigan (Neurotree) |
David A. Buchner | grad student | 2003 | University of Michigan (Neurotree) |
Stephen I. Levin | grad student | 2004 | University of Michigan (Neurotree) |
Bryan T. MacDonald | grad student | 2004 | University of Michigan (Neurotree) |
Valerie L. Drews | grad student | 2005 | University of Michigan (Neurotree) |
Cole J. Ferguson | grad student | 2011 | University of Michigan (Neurotree) |
Andrew P. Escayg | post-doc | (Cell Biology Tree) | |
David C. Kohrman | post-doc | (Neurotree) | |
Leslie Sprunger | post-doc | 1995-2000 | University of Michigan (Neurotree) |
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Publications
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Hill SF, Yu W, Ziobro J, et al. (2023) Long-term downregulation of the sodium channel gene Scn8a is therapeutic in mouse models of SCN8A epilepsy. Annals of Neurology |
Hill SF, Jafar-Nejad P, Rigo F, et al. (2023) Reduction of is therapeutic in mouse models of and epilepsy. Frontiers in Neuroscience. 17: 1282201 |
Cao X, Lenk GM, Meisler MH. (2023) Altered phenotypes due to genetic interaction between the mouse phosphoinositide biosynthesis genes Fig4 and Pip4k2c. G3 (Bethesda, Md.) |
Hill SF, Ziobro J, Jafar-Nejad P, et al. (2022) Genetic interaction between Scn8a and potassium channel genes Kcna1 and Kcnq2. Epilepsia |
Yu W, Mulligan MK, Williams RW, et al. (2022) Correction of the hypomorphic splice site variant in mouse strain C57BL/6J modifies the severity of encephalopathy. Hgg Advances. 3: 100064 |
Yu W, Smolen CE, Hill SF, et al. (2021) Spontaneous seizures and elevated seizure susceptibility in response to somatic mutation of sodium channel Scn8a in the mouse. Human Molecular Genetics |
Meisler MH, Hill SF, Yu W. (2021) Sodium channelopathies in neurodevelopmental disorders. Nature Reviews. Neuroscience |
Lenk GM, Jafar-Nejad P, Hill SF, et al. (2020) Scn8a antisense oligonucleotide is protective in mouse models of SCN8A Encephalopathy and Dravet Syndrome. Annals of Neurology |
Du J, Simmons S, Brunklaus A, et al. (2019) Differential excitatory vs inhibitory SCN expression at single cell level regulates brain sodium channel function in neurodevelopmental disorders. European Journal of Paediatric Neurology : Ejpn : Official Journal of the European Paediatric Neurology Society |
Meisler MH. (2019) SCN8A encephalopathy: Mechanisms and models. Epilepsia. 60: S86-S91 |