George Chaconas

Affiliations: 
Microbiology, Immunology, and Infectious Diseases University of Calgary, Calgary, Alberta, Canada 
Area:
Microbiology, Molecular Biology, Genetics
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"George Chaconas"
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Publications

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Castellanos M, Verhey TB, Goldstein M, et al. (2022) The Putative Endonuclease Activity of MutL Is Required for the Segmental Gene Conversion Events That Drive Antigenic Variation of the Lyme Disease Spirochete. Frontiers in Microbiology. 13: 888494
Lin YP, Tan X, Caine JA, et al. (2020) Strain-specific joint invasion and colonization by Lyme disease spirochetes is promoted by outer surface protein C. Plos Pathogens. 16: e1008516
Chaconas G, Castellanos M, Verhey TB. (2019) Changing of the guard: How the Lyme disease spirochete subverts the host immune response. The Journal of Biological Chemistry
Verhey TB, Castellanos M, Chaconas G. (2018) Antigenic variation in the Lyme spirochete: detailed functional assessment of recombinational switching at vlsE in the JD1 strain of Borrelia burgdorferi. Molecular Microbiology
Castellanos M, Verhey TB, Chaconas G. (2018) A Borrelia burgdorferi mini-vls system that undergoes antigenic switching in mice: investigation of the role of plasmid topology and the long inverted repeat. Molecular Microbiology
Verhey TB, Castellanos M, Chaconas G. (2018) Antigenic Variation in the Lyme Spirochete: Insights into Recombinational Switching with a Suggested Role for Error-Prone Repair. Cell Reports. 23: 2595-2605
Verhey TB, Castellanos M, Chaconas G. (2017) Analysis of recombinational switching at the antigenic variation locus of the Lyme spirochete using a novel PacBio sequencing pipeline. Molecular Microbiology
Kumar D, Ristow LC, Shi M, et al. (2015) Intravital Imaging of Vascular Transmigration by the Lyme Spirochete: Requirement for the Integrin Binding Residues of the B. burgdorferi P66 Protein. Plos Pathogens. 11: e1005333
Kobryn K, Chaconas G. (2014) Hairpin Telomere Resolvases. Microbiology Spectrum. 2
Lee WY, Sanz MJ, Wong CH, et al. (2014) Invariant natural killer T cells act as an extravascular cytotoxic barrier for joint-invading Lyme Borrelia. Proceedings of the National Academy of Sciences of the United States of America. 111: 13936-41
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