Bradley B. Brasher, Ph.D. - Publications
Affiliations: | 2000 | Harvard University, Cambridge, MA, United States |
Area:
GeneticsYear | Citation | Score | |||
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2019 | Teyra J, Singer AU, Schmitges FW, Jaynes P, Kit Leng Lui S, Polyak MJ, Fodil N, Krieger JR, Tong J, Schwerdtfeger C, Brasher BB, Ceccarelli DFJ, Moffat J, Sicheri F, Moran MF, et al. Structural and Functional Characterization of Ubiquitin Variant Inhibitors of USP15. Structure (London, England : 1993). PMID 30713027 DOI: 10.1016/J.Str.2019.01.002 | 0.428 | |||
2002 | Roumiantsev S, Shah NP, Gorre ME, Nicoll J, Brasher BB, Sawyers CL, Van Etten RA. Clinical resistance to the kinase inhibitor STI-571 in chronic myeloid leukemia by mutation of Tyr-253 in the Abl kinase domain P-loop. Proceedings of the National Academy of Sciences of the United States of America. 99: 10700-5. PMID 12149456 DOI: 10.1073/Pnas.162140299 | 0.6 | |||
2001 | Brasher BB, Roumiantsev S, Van Etten RA. Mutational analysis of the regulatory function of the c-Abl Src homology 3 domain. Oncogene. 20: 7744-52. PMID 11753652 DOI: 10.1038/Sj.Onc.1204978 | 0.619 | |||
2001 | Li S, Couvillon AD, Brasher BB, Van Etten RA. Tyrosine phosphorylation of Grb2 by Bcr/Abl and epidermal growth factor receptor: a novel regulatory mechanism for tyrosine kinase signaling. The Embo Journal. 20: 6793-804. PMID 11726515 DOI: 10.1093/Emboj/20.23.6793 | 0.603 | |||
2000 | Brasher BB, Van Etten RA. c-Abl has high intrinsic tyrosine kinase activity that is stimulated by mutation of the Src homology 3 domain and by autophosphorylation at two distinct regulatory tyrosines. The Journal of Biological Chemistry. 275: 35631-7. PMID 10964922 DOI: 10.1074/Jbc.M005401200 | 0.611 | |||
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