Ursula Storb

Affiliations: 
University of Chicago, Chicago, IL 
Area:
Molecular Biology, Cell Biology, Immunology
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"Ursula Storb"
Bio:

https://www.awis-chicago.org/community/scientist-of-the-month/march-2015-sotm-ursula-storb

Cross-listing: Cell Biology Tree

Children

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Linda B. Buck research assistant University of Washington
James R Hagman grad student 1983-1989 Chicago (Cell Biology Tree)
Charles Rudin grad student 1986-1991 Chicago (Cell Biology Tree)
Nayun Kim grad student 2000 Chicago (Cell Biology Tree)
Tianhe Sun grad student 2001 Chicago (Cell Biology Tree)
Simonne C. Longerich grad student 2006 Chicago (Cell Biology Tree)
Kristoffer G. Padjen grad student 2006 Chicago (Cell Biology Tree)
Atsushi Tanaka grad student 2009 Chicago (Cell Biology Tree)
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Publications

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Ratnam S, Bozek G, Payne C, et al. (2017) Ssm1b expression and function in germ cells of adult mice and in early embryos. Molecular Reproduction and Development
Ratnam S, Engler P, Bozek G, et al. (2014) Identification of Ssm1b, a novel modifier of DNA methylation, and its expression during mouse embryogenesis. Development (Cambridge, England). 141: 2024-34
Storb U. (2014) Why does somatic hypermutation by AID require transcription of its target genes? Advances in Immunology. 122: 253-77
Kodgire P, Mukkawar P, Ratnam S, et al. (2013) Changes in RNA polymerase II progression influence somatic hypermutation of Ig-related genes by AID. The Journal of Experimental Medicine. 210: 1481-92
Kodgire P, Mukkawar P, North JA, et al. (2012) Nucleosome stability dramatically impacts the targeting of somatic hypermutation. Molecular and Cellular Biology. 32: 2030-40
Ratnam S, Bozek G, Nicolae D, et al. (2010) The pattern of somatic hypermutation of Ig genes is altered when p53 is inactivated. Molecular Immunology. 47: 2611-8
Tanaka A, Shen HM, Ratnam S, et al. (2010) Attracting AID to targets of somatic hypermutation. The Journal of Experimental Medicine. 207: 405-15
Storb U, Shen HM, Nicolae D. (2009) Somatic hypermutation: processivity of the cytosine deaminase AID and error-free repair of the resulting uracils. Cell Cycle (Georgetown, Tex.). 8: 3097-101
Shen HM, Poirier MG, Allen MJ, et al. (2009) The activation-induced cytidine deaminase (AID) efficiently targets DNA in nucleosomes but only during transcription. The Journal of Experimental Medicine. 206: 1057-71
Shen HM, Bozek G, Pinkert CA, et al. (2008) Expression of AID transgene is regulated in activated B cells but not in resting B cells and kidney. Molecular Immunology. 45: 1883-92
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