Shu Zhang
Affiliations: | Neuroscience | University of British Columbia, Vancouver, Vancouver, BC, Canada |
Area:
Pten, NMDARs, StrokeGoogle:
"Shu Zhang"Mean distance: 16.09 (cluster 6) | S | N | B | C | P |
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Publications
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Girling KD, Demers MJ, Laine J, et al. (2017) Activation of caspase-6 and cleavage of caspase-6 substrates is an early event in NMDA receptor-mediated excitotoxicity. Journal of Neuroscience Research |
Dong Z, Han H, Li H, et al. (2015) Long-term potentiation decay and memory loss are mediated by AMPAR endocytosis. The Journal of Clinical Investigation. 125: 234-47 |
Lai TW, Zhang S, Wang YT. (2014) Excitotoxicity and stroke: identifying novel targets for neuroprotection. Progress in Neurobiology. 115: 157-88 |
Zhou N, Wang CH, Zhang S, et al. (2013) The GLRA1 missense mutation W170S associates lack of Zn2+ potentiation with human hyperekplexia. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 33: 17675-81 |
Zhang S, Taghibiglou C, Girling K, et al. (2013) Critical role of increased PTEN nuclear translocation in excitotoxic and ischemic neuronal injuries. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 33: 7997-8008 |
Demers MJ, Girling K, Zhang S, et al. (2013) Early activation of caspase-6 and cleavage of caspase-6 substrates in an in vitro model of stroke Autonomic Neuroscience: Basic and Clinical. 177: 48 |
Liu J, Fang PH, Hou Y, et al. (2010) The value of transtelephonic electrocardiogram monitoring system during the "Blanking Period" after ablation of atrial fibrillation. Journal of Electrocardiology. 43: 667-72 |
Taghibiglou C, Martin HG, Lai TW, et al. (2009) Role of NMDA receptor-dependent activation of SREBP1 in excitotoxic and ischemic neuronal injuries. Nature Medicine. 15: 1399-406 |